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Tumour necrosis factor enhances the interferon-gamma-induced class II MHC antigen expression on murine keratinocytes in vivo.

作者信息

Nakamura K, Tamaki K

机构信息

Department of Dermatology, Tokyo University Branch Hospital, University of Tokyo, Japan.

出版信息

Arch Dermatol Res. 1990;282(6):415-7. doi: 10.1007/BF00372095.

DOI:10.1007/BF00372095
PMID:2124437
Abstract
摘要

相似文献

1
Tumour necrosis factor enhances the interferon-gamma-induced class II MHC antigen expression on murine keratinocytes in vivo.肿瘤坏死因子增强体内小鼠角质形成细胞上γ干扰素诱导的II类主要组织相容性复合体抗原的表达。
Arch Dermatol Res. 1990;282(6):415-7. doi: 10.1007/BF00372095.
2
MHC class II antigen expression is not induced on murine epidermal keratinocytes by interferon-gamma alone or in combination with tumour necrosis factor-alpha.单独的γ干扰素或其与α肿瘤坏死因子联合使用时，不会诱导小鼠表皮角质形成细胞表达II类主要组织相容性复合体抗原。
Immunology. 1989 Jan;66(1):100-5.
3
Cytokine regulation of major histocompatibility complex antigen expression by human oral and skin keratinocytes.细胞因子对人口腔和皮肤角质形成细胞主要组织相容性复合体抗原表达的调控
Arch Oral Biol. 1996 Jun;41(6):533-8. doi: 10.1016/0003-9969(96)00026-x.
4
Schwann cells co-cultured with stimulated T cells and antigen express major histocompatibility complex (MHC) class II determinants without interferon-gamma pretreatment: synergistic effects of interferon-gamma and tumor necrosis factor on MHC class II induction.与活化的T细胞和抗原共培养的施万细胞在未进行γ干扰素预处理的情况下表达主要组织相容性复合体（MHC）II类决定簇：γ干扰素和肿瘤坏死因子对MHC II类诱导的协同作用。
Eur J Immunol. 1989 Jan;19(1):177-83. doi: 10.1002/eji.1830190128.
5
Class II transactivator-mediated regulation of major histocompatibility complex class II antigen expression is important for hematopoietic progenitor cell suppression by chemokines and iron-binding proteins.II类反式激活因子介导的主要组织相容性复合体II类抗原表达调控对于趋化因子和铁结合蛋白对造血祖细胞的抑制作用很重要。
Exp Hematol. 2006 Aug;34(8):1078-84. doi: 10.1016/j.exphem.2006.04.008.
6
Expression and modulation of major histocompatibility antigens on murine primary brain tumor in vitro.小鼠原发性脑肿瘤主要组织相容性抗原在体外的表达与调控
J Neurosurg. 1991 Dec;75(6):922-9. doi: 10.3171/jns.1991.75.6.0922.
7
Tumor necrosis factor alpha and interleukin-1 alpha inhibit through different pathways interferon-gamma-induced antigen presentation, processing and MHC class II surface expression on astrocytes, but not on microglia.肿瘤坏死因子α和白细胞介素-1α通过不同途径抑制γ干扰素诱导的星形胶质细胞而非小胶质细胞的抗原呈递、加工以及MHC II类分子的表面表达。
Cell Tissue Res. 2000 Jun;300(3):373-82. doi: 10.1007/s004410000222.
8
Regulation of MHC molecules on MBP positive oligodendrocytes in mice by IFN-gamma and TNF-alpha.干扰素-γ和肿瘤坏死因子-α对小鼠MBP阳性少突胶质细胞上MHC分子的调控
Neurosci Lett. 1991 Feb 11;123(1):45-8. doi: 10.1016/0304-3940(91)90154-l.
9
Interferon-gamma-stimulated human keratinocytes express the genes necessary for the production of peptide-loaded MHC class II molecules.
J Invest Dermatol. 1998 Feb;110(2):138-42. doi: 10.1046/j.1523-1747.1998.00098.x.
10
Regulation of MHC class II antigen expression. Opposing effects of tumor necrosis factor-alpha on IFN-gamma-induced HLA-DR and Ia expression depends on the maturation and differentiation stage of the cell.MHC II类抗原表达的调控。肿瘤坏死因子-α对干扰素-γ诱导的HLA-DR和Ia表达的相反作用取决于细胞的成熟和分化阶段。
J Immunol. 1991 Feb 1;146(3):899-905.

引用本文的文献

1
Immunohistological and immunoelectron microscopic identification of TNF alpha in normal human and murine epidermis.正常人和小鼠表皮中肿瘤坏死因子α的免疫组织学和免疫电子显微镜鉴定
Arch Dermatol Res. 1992;284(3):154-8. doi: 10.1007/BF00372709.

本文引用的文献

1
MHC class II antigen expression is not induced on murine epidermal keratinocytes by interferon-gamma alone or in combination with tumour necrosis factor-alpha.单独的γ干扰素或其与α肿瘤坏死因子联合使用时，不会诱导小鼠表皮角质形成细胞表达II类主要组织相容性复合体抗原。
Immunology. 1989 Jan;66(1):100-5.
2
Recombinant gamma interferon induces HLA-DR expression on cultured human keratinocytes.重组γ干扰素可诱导培养的人角质形成细胞表达HLA - DR。
J Invest Dermatol. 1984 Aug;83(2):88-90. doi: 10.1111/1523-1747.ep12262597.
3
Shock and tissue injury induced by recombinant human cachectin.
重组人恶病质素诱导的休克和组织损伤
Science. 1986 Oct 24;234(4775):470-4. doi: 10.1126/science.3764421.
4
Immunohistological detection of interleukin I-like molecules and tumour necrosis factor in human epidermis before and after UVB-irradiation in vivo.
Br J Dermatol. 1988 Mar;118(3):369-76. doi: 10.1111/j.1365-2133.1988.tb02430.x.
5
Human epidermal cells synthesize HLA-DR alloantigens in vitro upon stimulation with gamma-interferon.人表皮细胞在γ干扰素刺激下可在体外合成HLA - DR同种抗原。
J Invest Dermatol. 1985 Jul;85(1):16-9. doi: 10.1111/1523-1747.ep12274511.
6
Induction and functional characterization of class II MHC (Ia) antigens on murine keratinocytes.小鼠角质形成细胞上II类主要组织相容性复合体（Ia）抗原的诱导及功能特性
J Immunol. 1988 May 1;140(9):2956-63.
7
Immunoregulatory activity of recombinant human tumor necrosis factor (TNF)-alpha: induction of TNF receptors on human T cells and TNF-alpha-mediated enhancement of T cell responses.重组人肿瘤坏死因子（TNF）-α的免疫调节活性：诱导人T细胞上的TNF受体以及TNF-α介导的T细胞反应增强。
J Immunol. 1987 Mar 15;138(6):1786-90.
8
Cachectin and tumour necrosis factor as two sides of the same biological coin.恶病质素与肿瘤坏死因子是同一枚生物硬币的两面。
Nature. 1986;320(6063):584-8. doi: 10.1038/320584a0.
9
Differential expression of Ia by murine keratinocytes and gut epithelium in response to recombinant gamma-interferon.小鼠角质形成细胞和肠道上皮细胞对重组γ干扰素反应时Ia的差异表达
J Invest Dermatol. 1989 Jul;93(1):33-9. doi: 10.1111/1523-1747.ep12277340.
10
IFN-mediated induction of MHC antigen expression on human keratinocytes and its influence on in vitro alloimmune responses.干扰素介导的人类角质形成细胞上主要组织相容性复合体抗原表达的诱导及其对体外同种免疫反应的影响。
J Immunol. 1988 Apr 15;140(8):2556-64.