• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Role of tumor necrosis factor and colony-stimulating factors in phagocyte function against Candida albicans.

作者信息

Djeu J Y

机构信息

University of South Florida College of Medicine, Department of Medical Microbiology and Immunology, Tampa 33612.

出版信息

Diagn Microbiol Infect Dis. 1990 Sep-Oct;13(5):383-6. doi: 10.1016/0732-8893(90)90007-i.

DOI:10.1016/0732-8893(90)90007-i
PMID:2126496
Abstract
摘要

相似文献

1
Role of tumor necrosis factor and colony-stimulating factors in phagocyte function against Candida albicans.
Diagn Microbiol Infect Dis. 1990 Sep-Oct;13(5):383-6. doi: 10.1016/0732-8893(90)90007-i.
2
Colony-stimulating factor enhancement of myeloid effector cell cytotoxicity towards neuroectodermal tumour cells.集落刺激因子增强髓样效应细胞对神经外胚层肿瘤细胞的细胞毒性。
Br J Haematol. 1993 Apr;83(4):545-53. doi: 10.1111/j.1365-2141.1993.tb04689.x.
3
Granulocyte-macrophage colony-stimulating factor augments human monocyte fungicidal activity for Candida albicans.粒细胞巨噬细胞集落刺激因子增强人单核细胞对白色念珠菌的杀菌活性。
J Infect Dis. 1990 May;161(5):999-1005. doi: 10.1093/infdis/161.5.999.
4
Activation of human eosinophil and neutrophil functions by haematopoietic growth factors: comparisons of IL-1, IL-3, IL-5 and GM-CSF.造血生长因子对人嗜酸性粒细胞和中性粒细胞功能的激活作用：白细胞介素-1、白细胞介素-3、白细胞介素-5和粒细胞巨噬细胞集落刺激因子的比较
Br J Haematol. 1992 Feb;80(2):137-43. doi: 10.1111/j.1365-2141.1992.tb08890.x.
5
Enhancement of human monocyte function against Candida albicans by the colony-stimulating factors (CSF): IL-3, granulocyte-macrophage-CSF, and macrophage-CSF.集落刺激因子（CSF）：白细胞介素-3、粒细胞-巨噬细胞集落刺激因子和巨噬细胞集落刺激因子对人类单核细胞抗白色念珠菌功能的增强作用。
J Immunol. 1989 Jul 15;143(2):671-7.
6
Augmentation of human macrophage candidacidal capacity by recombinant human myeloperoxidase and granulocyte-macrophage colony-stimulating factor.重组人髓过氧化物酶和粒细胞巨噬细胞集落刺激因子增强人巨噬细胞对念珠菌的杀伤能力
Infect Immun. 1998 Jun;66(6):2750-4. doi: 10.1128/IAI.66.6.2750-2754.1998.
7
Regulation by interleukin-3 of human monocyte pro-inflammatory mediators. Similarities with granulocyte-macrophage colony-stimulating factor.白细胞介素-3对人单核细胞促炎介质的调节作用。与粒细胞-巨噬细胞集落刺激因子的相似性。
Immunology. 1990 Sep;71(1):76-82.
8
Effect of granulocyte-macrophage colony-stimulating factor on candidacidal activity of neutrophils, monocytes or monocyte-derived macrophages and synergy with fluconazole.粒细胞巨噬细胞集落刺激因子对中性粒细胞、单核细胞或单核细胞衍生巨噬细胞杀念珠菌活性的影响以及与氟康唑的协同作用。
J Med Microbiol. 1998 Apr;47(4):359-63. doi: 10.1099/00222615-47-4-359.
9
Response of human polymorphonuclear leukocytes and monocytes to Trichosporon beigelii: host defense against an emerging opportunistic pathogen.人类多形核白细胞和单核细胞对白吉利丝孢酵母的反应：针对一种新出现的机会性病原体的宿主防御。
J Infect Dis. 1994 Dec;170(6):1557-65. doi: 10.1093/infdis/170.6.1557.
10
Enhanced phagocytosis and intracellular killing of Candida albicans by GM-CSF-activated human neutrophils.粒细胞巨噬细胞集落刺激因子激活的人中性粒细胞增强对白色念珠菌的吞噬作用和细胞内杀伤作用。
J Med Vet Mycol. 1992;30(6):433-41.

引用本文的文献

1
The RIG-I-like helicase receptor MDA5 (IFIH1) is involved in the host defense against Candida infections.视黄酸诱导基因I样解旋酶受体黑色素瘤分化相关基因5（IFIH1）参与宿主对念珠菌感染的防御。
Eur J Clin Microbiol Infect Dis. 2015 May;34(5):963-974. doi: 10.1007/s10096-014-2309-2. Epub 2015 Jan 13.
2
Serial passaging of Candida albicans in systemic murine infection suggests that the wild type strain SC5314 is well adapted to the murine kidney.系统性真菌感染的白色念珠菌连续传代提示野生型菌株 SC5314 能很好地适应鼠肾。
PLoS One. 2013 May 30;8(5):e64482. doi: 10.1371/journal.pone.0064482. Print 2013.
3
Virulence attenuation of Candida albicans genetic variants isolated from a patient with a recurrent bloodstream infection.
从复发性血流感染患者中分离出的白色念珠菌遗传变异株的毒力减弱。
PLoS One. 2010 Apr 13;5(4):e10155. doi: 10.1371/journal.pone.0010155.
4
Endoplasmic reticulum alpha-glycosidases of Candida albicans are required for N glycosylation, cell wall integrity, and normal host-fungus interaction.白色念珠菌的内质网α-糖苷酶是N-糖基化、细胞壁完整性和正常宿主-真菌相互作用所必需的。
Eukaryot Cell. 2007 Dec;6(12):2184-93. doi: 10.1128/EC.00350-07. Epub 2007 Oct 12.
5
Immune sensing of Candida albicans requires cooperative recognition of mannans and glucans by lectin and Toll-like receptors.白色念珠菌的免疫感知需要凝集素和Toll样受体对甘露聚糖和葡聚糖的协同识别。
J Clin Invest. 2006 Jun;116(6):1642-50. doi: 10.1172/JCI27114. Epub 2006 May 18.
6
Comparison of pathogenesis and host immune responses to Candida glabrata and Candida albicans in systemically infected immunocompetent mice.在全身感染的免疫功能正常小鼠中，光滑念珠菌和白色念珠菌的发病机制及宿主免疫反应比较
Infect Immun. 2001 Aug;69(8):5046-55. doi: 10.1128/IAI.69.8.5046-5055.2001.
7
Intravenous injection of Candida-derived mannan results in elevated tumor necrosis factor alpha levels in serum.静脉注射念珠菌来源的甘露聚糖会导致血清中肿瘤坏死因子α水平升高。
Infect Immun. 1996 Nov;64(11):4561-6. doi: 10.1128/iai.64.11.4561-4566.1996.
8
Tumor necrosis factor alpha has a protective role in a murine model of systemic candidiasis.肿瘤坏死因子α在系统性念珠菌病小鼠模型中具有保护作用。
Infect Immun. 1994 Jul;62(7):2761-72. doi: 10.1128/iai.62.7.2761-2772.1994.