粗糙脉孢菌通过热休克及其他诱导过氧化物酶产生的应激反应形成耐热性。
Development of thermotolerance in Neurospora crassa by heat shock and other stresses eliciting peroxidase induction.
作者信息
Kapoor M, Sreenivasan G M, Goel N, Lewis J
机构信息
Department of Biological Sciences, University of Calgary, Alberta, Canada.
出版信息
J Bacteriol. 1990 May;172(5):2798-801. doi: 10.1128/jb.172.5.2798-2801.1990.
Abstract
Hyperthermia, CdCl2, sodium arsenite, and H2O2 led to the rapid appearance of high levels of peroxidase in Neurospora crassa cultures and induced tolerance toward normally lethal temperatures in 60-h-old colonies. Intracellular superoxide dismutase levels did not correlate with the development of thermotolerance.
摘要
高温、氯化镉、亚砷酸钠和过氧化氢导致粗糙脉孢菌培养物中迅速出现高水平的过氧化物酶，并使60小时龄的菌落对正常致死温度产生耐受性。细胞内超氧化物歧化酶水平与耐热性的发展无关。
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