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大肠杆菌麦芽糖结合蛋白的折叠特性在体外影响其与SecB的相互作用。

The folding properties of the Escherichia coli maltose-binding protein influence its interaction with SecB in vitro.

作者信息

Weiss J B, Bassford P J

机构信息

Department of Microbiology and Immunology, School of Medicine, University of North Carolina, Chapel Hill 27599-7290.

出版信息

J Bacteriol. 1990 Jun;172(6):3023-9. doi: 10.1128/jb.172.6.3023-3029.1990.

Abstract

It has been proposed that the cytoplasmic SecB protein functions as a component of the Escherichia coli protein export machinery by serving as an antifolding factor that retards folding of the precursor maltose-binding protein (preMBP) into a translocation-incompetent form. In this study, it was found that SecB directly interacts with wild-type preMBP and various mutationally altered MBP species synthesized in vitro to form a SecB-MBP complex that can be precipitated with anti-SecB serum. The association of SecB with wild-type preMBP was relatively unstable; such a complex was formed only when SecB was present cotranslationally or after denaturation of previously synthesized preMBP and was detected with only low efficiency. In marked contrast, MBP species that were defective in the ability to assume the stable conformation of wild-type preMBP or that exhibited significantly slower folding kinetics formed much more stable complexes with SecB. In one case, we demonstrated that SecB did not need to be present cotranslationally for complex formation to occur. Formation of a complex between SecB and MBP was clearly not dependent on the MBP signal peptide. However, we were unable to detect complex formation between SecB and MBP lacking virtually the entire signal peptide but having a completely intact mature moiety. This MBP species folded at a rate considerably faster than that of wild-type preMBP. The propensity of this mutant protein to assume the native conformation of mature MBP apparently precludes a stable association with SecB, whereas an MBP species lacking a signal peptide but exhibiting altered folding properties did form a complex with SecB that could be precipitated with anti-SecB serum.

摘要

有人提出,细胞质中的SecB蛋白作为大肠杆菌蛋白质输出机制的一个组成部分,充当抗折叠因子,减缓前体麦芽糖结合蛋白(preMBP)折叠成无法转运的形式。在本研究中,发现SecB与体外合成的野生型preMBP及各种突变的MBP物种直接相互作用,形成一种可被抗SecB血清沉淀的SecB-MBP复合物。SecB与野生型preMBP的结合相对不稳定;只有当SecB在共翻译时存在,或在先前合成的preMBP变性后存在时,才会形成这样的复合物,且检测效率很低。与之形成鲜明对比的是,那些在形成野生型preMBP稳定构象的能力上有缺陷,或折叠动力学明显较慢的MBP物种,与SecB形成的复合物要稳定得多。在一个案例中,我们证明复合物形成时SecB不需要共翻译存在。SecB和MBP之间复合物的形成显然不依赖于MBP信号肽。然而,我们无法检测到SecB与几乎缺失整个信号肽但成熟部分完全完整的MBP之间的复合物形成。这种MBP物种的折叠速度比野生型preMBP快得多。这种突变蛋白呈现成熟MBP天然构象的倾向显然排除了与SecB的稳定结合,而一种缺失信号肽但折叠特性改变的MBP物种确实与SecB形成了一种可被抗SecB血清沉淀的复合物。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/70c8/209103/2def9cfde23f/jbacter00160-0219-a.jpg

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