Human Performance Laboratory, University of Technology Sydney, Lindfield, Australia.
Appl Physiol Nutr Metab. 2012 Apr;37(2):315-22. doi: 10.1139/h2012-001. Epub 2012 Mar 27.
This study examined the effect of training load on running performance and plasma markers of anaerobic metabolism, muscle damage, and inflammation during a simulated team sport match performance. Seven team sport athletes (maximal oxygen uptake, 47.6 ± 4.2 mL·kg(-1)·min(-1)) completed a 60-min simulated team sport match before and after either 4 days of HIGH or LOW training loads. Venous blood samples were taken pre-match, immediately post-match, and 2 h post-match for interlukin-6, monocyte chemoattractant protein-1 (MCP-1), creatine kinase (CK), lactate dehydrogenase, C-reactive protein, xanthine oxidase (XO), and hypoxanthine. Following HIGH training load, sprint velocity decreased (p < 0.001) and total distance covered was reduced (HIGH 5495 ± 670 m, LOW 5608 ± 674 m, p = 0.02) was observed during the simulated match protocol compared with the LOW match simulation. Decreased performance capacity was accompanied by a significant increase in serum CK concentration (HIGH 290 ± 62 U·L(-1), LOW 199 ± 33 U·L(-1), p = 0.005). The HIGH training also resulted in a decreased post-match hypoxanthine and MCP-1 and an increase in XO concentration 2 h post-match. Four days of increased training load reduced running performance during the match simulation and altered the metabolic and inflammatory response to high-intensity intermittent exercise.
本研究考察了训练负荷对模拟团队运动比赛表现中跑步表现和无氧代谢、肌肉损伤和炎症的血浆标志物的影响。7 名团队运动运动员（最大摄氧量 47.6 ± 4.2 mL·kg(-1)·min(-1)）在接受 4 天高或低训练负荷前后完成了 60 分钟的模拟团队运动比赛。在比赛前、比赛后立即和比赛后 2 小时采集静脉血样，用于检测白细胞介素-6、单核细胞趋化蛋白-1（MCP-1）、肌酸激酶（CK）、乳酸脱氢酶、C 反应蛋白、黄嘌呤氧化酶（XO）和次黄嘌呤。与低负荷比赛模拟相比，高负荷训练后，冲刺速度下降（p < 0.001），模拟比赛中总距离减少（高负荷 5495 ± 670 m，低负荷 5608 ± 674 m，p = 0.02）。运动能力下降伴随着血清 CK 浓度的显著增加（高负荷 290 ± 62 U·L(-1)，低负荷 199 ± 33 U·L(-1)，p = 0.005）。高负荷训练还导致比赛后次黄嘌呤和 MCP-1 减少，XO 浓度在比赛后 2 小时增加。4 天的增加训练负荷降低了比赛模拟中的跑步表现，并改变了高强度间歇运动的代谢和炎症反应。
