New York University School of Medicine, Department of Psychiatry, 215 Lexington Ave., 15th Floor, New York, NY 10016, USA.
Addict Behav. 2013 Jul;38(7):2361-8. doi: 10.1016/j.addbeh.2013.03.008. Epub 2013 Mar 21.
While smoking is a major cause of mortality and morbidity, and maternal smoking is a risk factor for smoking among their offspring, the mechanisms involved in the intergenerational transmission of smoking are not well understood. This study examines the pathways from maternal and adolescent child factors, and the parent-child relationship, to smoking among the adult offspring, approximately 25 years later. Data for the present analysis were based on time waves 2 (T2; 1983) and 7 (T7; 2007-2009) of an on-going study of a community sample of mothers and their children. Offspring and mother X¯ ages were 14.1 and 40.0 years, respectively, at T2, and 36.6 and 65.0 years, respectively, at T7. At T2, trained interviewers administered individual structured interviews. Psychosocial questionnaires were self-administered at T7. Structural equation modeling (SEM) was used to analyze the interrelationships among maternal and offspring attributes (T2 and T7). SEM results indicated a satisfactory model fit (RMSEA=0.052; CFI=0.91; SRMR=0.057), and confirmed hypothesized pathways. One pathway linked maternal maladaptive attributes (T2) to the mother-adolescent child attachment relationship (T2), which was associated with the offspring's maladaptive attributes over time (T2 to T7), which then predicted the adult offspring's smoking (T7). Other pathways highlighted the stability of maternal smoking, the continuity of maladaptive attributes, and less offspring educational attainment as predictors of offspring smoking at T7. Findings suggest the importance of early interventions to treat maternal smoking, maternal and offspring maladaptive attributes, and the mother-child relationship in order to reduce risk factors for the intergenerational transmission of smoking behavior. Interventions which enhance educational success should also prove effective in reducing smoking.
虽然吸烟是导致死亡率和发病率的主要原因,而且母亲吸烟是其子女吸烟的一个危险因素,但代际间吸烟行为传递的机制尚不清楚。本研究考察了从母亲和青少年子女因素,以及亲子关系到成年子女吸烟的途径,大约 25 年后。本分析的数据基于一项对母亲及其子女进行的社区样本进行的正在进行的研究的时间波 2(T2;1983 年)和 7(T7;2007-2009 年)。在 T2 时,子女和母亲的 X¯年龄分别为 14.1 岁和 40.0 岁,在 T7 时,分别为 36.6 岁和 65.0 岁。在 T2 时,经过培训的访谈者进行了个人结构化访谈。在 T7 时,心理社会问卷由自己填写。结构方程模型(SEM)用于分析母亲和子女属性(T2 和 T7)之间的相互关系。SEM 结果表明模型拟合良好(RMSEA=0.052;CFI=0.91;SRMR=0.057),并证实了假设的途径。一条途径将母亲适应不良的属性(T2)与母子依恋关系(T2)联系起来,母子依恋关系与子女的适应不良属性随时间的变化(T2 到 T7)有关,而这些属性又预测了成年子女的吸烟行为(T7)。其他途径强调了母亲吸烟的稳定性、适应不良属性的连续性以及子女受教育程度较低作为 T7 时子女吸烟的预测因素的重要性。研究结果表明,为了减少吸烟行为代际传递的危险因素,需要早期干预来治疗母亲吸烟、母亲和子女的适应不良属性以及母子关系。增强教育成功的干预措施也应该有助于减少吸烟。