Laskova I L
Zh Mikrobiol Epidemiol Immunobiol. 1990 Mar(3):68-73.
Injection of live staphylococcal culture into mice induces the development of delayed hypersensitivity (DH) to microbial cells and suppresses the development of humoral immune response (HIR) to sheep red blood cells (SRBC). Physical loading of low intensity normalizes staphylococcus-suppressed HIR to SRBC, but produces no effect on staphylococcus-induced DH. Highly intensive physical loading suppresses the development of DH and enhances the suppressive effect of staphylococci on SRBC-induced HIR. The infection of animals with staphylococci induces the secretion of immunosuppressive factors by spleen cells. Physical loading of low intensity does not suppress the staphylococcus-induced secretion of suppressive factors by spleen cells, but induces the secretion of helper factors by these cells. Highly intensive physical loading enhances the secretion of immunosuppressive factors by spleen cells after infection with staphylococci.
将活的葡萄球菌培养物注射到小鼠体内会诱导对微生物细胞的迟发型超敏反应（DH）的发展，并抑制对绵羊红细胞（SRBC）的体液免疫反应（HIR）的发展。低强度的体力负荷可使葡萄球菌抑制的对SRBC的HIR恢复正常，但对葡萄球菌诱导的DH没有影响。高强度的体力负荷会抑制DH的发展，并增强葡萄球菌对SRBC诱导的HIR的抑制作用。用葡萄球菌感染动物会诱导脾细胞分泌免疫抑制因子。低强度的体力负荷不会抑制葡萄球菌诱导的脾细胞分泌抑制因子，但会诱导这些细胞分泌辅助因子。高强度的体力负荷会增强葡萄球菌感染后脾细胞免疫抑制因子的分泌。
