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The mechanisms of acrylamide axonopathy.

作者信息

Miller M S, Spencer P S

出版信息

Annu Rev Pharmacol Toxicol. 1985;25:643-66. doi: 10.1146/annurev.pa.25.040185.003235.

DOI:10.1146/annurev.pa.25.040185.003235
PMID:2408560
Abstract
摘要

相似文献

1
The mechanisms of acrylamide axonopathy.
Annu Rev Pharmacol Toxicol. 1985;25:643-66. doi: 10.1146/annurev.pa.25.040185.003235.
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Morphological studies of toxic distal axonopathy.
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6
Progressive deficits in retrograde axon transport precede degeneration of motor axons in acrylamide neuropathy.在丙烯酰胺神经病中，逆行轴突运输的进行性缺陷先于运动轴突的退化。
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Enhancement of sensitivity to acrylamide after nerve ligature.神经结扎后对丙烯酰胺敏感性的增强。
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8
[Impairment of rapid axonal transport and concomitant anomaly of smooth endoplasmic reticulum in acrylamide induced neuropathy].
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Toxic axonal degeneration occurs independent of neurofilament accumulation.
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How does acrylamide perturb axon transport and induce nerve fiber degeneration? Commentary on forum position paper.丙烯酰胺如何扰乱轴突运输并导致神经纤维变性？对论坛立场文件的评论。
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Sertoli cell processes have axoplasmic features: an ordered microtubule distribution and an abundant high molecular weight microtubule-associated protein (cytoplasmic dynein).支持细胞的突起具有轴浆特征：微管分布有序，且有丰富的高分子量微管相关蛋白（胞质动力蛋白）。
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