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Further evidence for a role of S100B in mood disorders: a human gene expression mega-analysis.

作者信息

Schroeter Matthias L, Steiner Johann, Schönknecht Peter, Mueller Karsten

机构信息

Max Planck Institute for Human Cognitive and Brain Sciences, Stephanstr. 1A, 04103 Leipzig, Germany; Day Clinic of Cognitive Neurology, University of Leipzig, Leipzig, Germany; LIFE - Leipzig Research Center for Civilization Diseases, University of Leipzig, Germany.

Department of Psychiatry, University of Magdeburg, Germany.

出版信息

J Psychiatr Res. 2014 Jun;53:84-6. doi: 10.1016/j.jpsychires.2014.02.021. Epub 2014 Mar 1.

DOI:10.1016/j.jpsychires.2014.02.021
PMID:24629352
Abstract
摘要

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Further evidence for a role of S100B in mood disorders: a human gene expression mega-analysis.S100B在情绪障碍中作用的进一步证据:一项人类基因表达荟萃分析。
J Psychiatr Res. 2014 Jun;53:84-6. doi: 10.1016/j.jpsychires.2014.02.021. Epub 2014 Mar 1.
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S100B-immunopositive astrocytes and oligodendrocytes in the hippocampus are differentially afflicted in unipolar and bipolar depression: a postmortem study.在单相和双相抑郁症中,海马体中 S100B 免疫阳性星形胶质细胞和少突胶质细胞受到不同程度的影响:一项尸检研究。
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Serum S100B represents a new biomarker for mood disorders.血清 S100B 是一种新的情绪障碍生物标志物。
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The role of S100B protein as a potential marker in affective disorders.S100B蛋白作为情感障碍潜在标志物的作用。
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The role of S100B in the interaction between adipocytes and macrophages.S100B 在脂肪细胞与巨噬细胞相互作用中的作用。
Obesity (Silver Spring). 2014 Feb;22(2):371-9. doi: 10.1002/oby.20532. Epub 2013 Aug 19.
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S100B overexpression increases behavioral and neural plasticity in response to the social environment during adolescence.S100B 过表达增加了青春期对社会环境的行为和神经可塑性。
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The expression of endothelial barrier antigen (EBA) and S100B in the rat parietal cortex following brain irradiation.脑照射后大鼠顶叶皮质中内皮屏障抗原(EBA)和S100B的表达。
Brain Res. 2014 Apr 16;1558:84-9. doi: 10.1016/j.brainres.2014.02.032. Epub 2014 Feb 22.
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S100B and schizophrenia.S100B 与精神分裂症。
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S100B and brain derived neurotrophic factor in monozygotic twins with, at risk of and without affective disorders.S100B 和脑源性神经营养因子在有、有风险和没有情感障碍的同卵双胞胎中的表达。
J Affect Disord. 2020 Sep 1;274:726-732. doi: 10.1016/j.jad.2020.05.015. Epub 2020 May 25.

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Increased Serum NSE and S100B Indicate Neuronal and Glial Alterations in Subjects Under 71 Years With Mild Neurocognitive Disorder/Mild Cognitive Impairment.
血清NSE和S100B升高表明71岁以下患有轻度神经认知障碍/轻度认知功能损害的受试者存在神经元和神经胶质改变。
Front Cell Neurosci. 2022 Jul 14;16:788150. doi: 10.3389/fncel.2022.788150. eCollection 2022.
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Reduced density of glutamine synthetase immunoreactive astrocytes in different cortical areas in major depression but not in bipolar I disorder.重度抑郁症患者不同皮质区域中谷氨酰胺合成酶免疫反应性星形胶质细胞密度降低,但双相I型障碍患者并非如此。
Front Cell Neurosci. 2015 Aug 10;9:273. doi: 10.3389/fncel.2015.00273. eCollection 2015.
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