Ricardo Yukio Asano, Universidade Mogi das Cruzes, Center of Health Sciences, Mogi das Cruzes 08770-490, Brazil.
World J Diabetes. 2014 Oct 15;5(5):659-65. doi: 10.4239/wjd.v5.i5.659.
The literature has shown the efficiency of exercise in the control of type 2 diabetes (T2D), being suggested as one of the best kinds of non-pharmacological treatments for its population. Thus, the scientific production related to this phenomenon has growing exponentially. However, despite its advances, still there is a lack of studies that have carried out a review on the acute effects of physical exercise on metabolic and hemodynamic markers and possible control mechanisms of these indicators in individuals with T2D, not to mention that in a related way, these themes have been very little studied today. Therefore, the aim of this study was to organize and analyze the current scientific production about the acute effects of physical exercise on metabolic and hemodynamic markers and possible control mechanisms of these indicators in T2D individuals. For such, a research with the following keywords was performed: -exercise; diabetes and post-exercise hypotension; diabetes and excess post-exercise oxygen consumption; diabetes and acute effects in PUBMED, SCIELO and HIGHWIRE databases. From the analyzed studies, it is possible to conclude that, a single exercise session can promote an increase in the bioavailability of nitric oxide and elicit decreases in postexercise blood pressure. Furthermore, the metabolic stress from physical exercise can increase the oxidation of carbohydrate during the exercise and keep it, in high levels, the post exercise consumption of O², this phenomenon increases the rate of fat oxidation during recovery periods after exercise, improves glucose tolerance and insulin sensitivity and reduces glycemia between 2-72 h, which seems to be dependent on the exercise intensity and duration of the effort.
文献表明,运动在控制 2 型糖尿病(T2D)方面具有高效性,被认为是针对该人群的最佳非药物治疗方法之一。因此,与这种现象相关的科学研究呈指数级增长。然而,尽管取得了这些进展,但仍缺乏对运动对 T2D 个体代谢和血液动力学标志物的急性影响以及这些指标的可能控制机制进行综述的研究,更不用说这些主题在相关方面目前研究得很少。因此,本研究旨在组织和分析目前关于运动对 T2D 个体代谢和血液动力学标志物的急性影响以及这些指标的可能控制机制的科学研究。为此,在 PUBMED、SCIELO 和 HIGHWIRE 数据库中使用了以下关键词进行了一项研究:-exercise; diabetes and post-exercise hypotension; diabetes and excess post-exercise oxygen consumption; diabetes and acute effects。从分析的研究中,可以得出结论,单次运动可以促进一氧化氮的生物利用度增加,并降低运动后的血压。此外,运动的代谢应激可以增加运动期间碳水化合物的氧化,并在运动后保持高水平的 O²消耗,这种现象增加了运动后恢复期脂肪的氧化率,提高了葡萄糖耐量和胰岛素敏感性,并降低了 2-72 小时之间的血糖水平,这似乎取决于运动强度和运动持续时间。