与腺病毒感染及腺病毒介导的自然杀伤缺陷相关的斯蒂尔病
Still's disease associated with adenovirus infection and defect in adenovirus directed natural killing.
作者信息
Luder A S, Naphtali V, Ben Porat E, Lahat N
机构信息
Department of Pediatrics, Carmel Hospital (Faculty of Medicine, Technion-Israel Institute of Technology), Haifa.
出版信息
Ann Rheum Dis. 1989 Sep;48(9):781-6. doi: 10.1136/ard.48.9.781.
Abstract
Low natural killer (NK) activity towards adenovirus infected fibroblasts was detected in the peripheral blood of a child with Still's disease and was not normalised by the addition of interferon alfa or interleukin 2. NK cytotoxicity directed at K 562 target cells or polio infected fibroblasts was normal. This specific NK deficiency might have contributed to the development of the child's Still's disease.
摘要
在一名斯蒂尔病患儿的外周血中检测到对腺病毒感染的成纤维细胞的自然杀伤（NK）活性较低，并且添加干扰素α或白细胞介素2后并未使其恢复正常。针对K 562靶细胞或脊髓灰质炎感染的成纤维细胞的NK细胞毒性是正常的。这种特异性NK细胞缺陷可能促成了该患儿斯蒂尔病的发展。
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