Predicting Risk at the End of the End: Telomere G-tail as a Biomarker.
作者信息
Seimiya Hiroyuki
机构信息
Division of Molecular Biotherapy, Cancer Chemotherapy Center, Japanese Foundation for Cancer Research, Tokyo 135-8550, Japan.
出版信息
EBioMedicine. 2015 Jul 4;2(8):804-5. doi: 10.1016/j.ebiom.2015.07.006. eCollection 2015 Aug.
Abstract
摘要
相似文献
1
Predicting Risk at the End of the End: Telomere G-tail as a Biomarker.预测终点的风险：端粒G尾作为一种生物标志物。
EBioMedicine. 2015 Jul 4;2(8):804-5. doi: 10.1016/j.ebiom.2015.07.006. eCollection 2015 Aug.
2
Telomere length inheritance and aging.端粒长度遗传与衰老。
Mech Ageing Dev. 2008 Jan-Feb;129(1-2):17-26. doi: 10.1016/j.mad.2007.10.009. Epub 2007 Oct 30.
3
Altered nuclear functions in progeroid syndromes: a paradigm for aging research.早衰综合征中的核功能改变：衰老研究的范例
ScientificWorldJournal. 2009 Dec 16;9:1449-62. doi: 10.1100/tsw.2009.159.
4
Unraveling the pathogenesis of Hoyeraal-Hreidarsson syndrome, a complex telomere biology disorder.解析霍耶拉尔-赫雷达尔松综合征的发病机制，一种复杂的端粒生物学障碍。
Br J Haematol. 2015 Aug;170(4):457-71. doi: 10.1111/bjh.13442. Epub 2015 May 4.
5
[Telomere and telomerase in age related diseases].
Seikagaku. 2006 Mar;78(3):230-41.
6
Mechanisms of cardiovascular disease in accelerated aging syndromes.加速衰老综合征中心血管疾病的机制。
Circ Res. 2007 Jul 6;101(1):13-26. doi: 10.1161/CIRCRESAHA.107.153692.
7
The role of telomeres and vitamin D in cellular aging and age-related diseases.端粒和维生素 D 在细胞衰老和与年龄相关的疾病中的作用。
Clin Chem Lab Med. 2015 Oct;53(11):1661-78. doi: 10.1515/cclm-2014-1184.
8
Dyskeratosis congenita mutations in dyskerin SUMOylation consensus sites lead to impaired telomerase RNA accumulation and telomere defects.先天性角化不良突变导致核仁素 SUMOylation 保守位点缺陷，从而导致端粒酶 RNA 积累减少和端粒缺陷。
Hum Mol Genet. 2013 Sep 1;22(17):3498-507. doi: 10.1093/hmg/ddt204. Epub 2013 May 8.
9
A comparison of adult and childhood progerias: Werner syndrome and Hutchinson-Gilford progeria syndrome.
Adv Exp Med Biol. 1985;190:229-44. doi: 10.1007/978-1-4684-7853-2_10.
10
A hypothesis that aging results from defects in genetically produced proteins.
Med Hypotheses. 1997 Jun;48(6):531-3. doi: 10.1016/s0306-9877(97)90125-0.
引用本文的文献
1
Does the Protective Effect of Zinc on Telomere Length Depend on the Presence of Hypertension or Type 2 Diabetes? Results from the Iwaki Health Promotion Project, Japan.锌对端粒长度的保护作用是否取决于高血压或 2 型糖尿病的存在？来自日本磐城健康促进计划的结果。
Nutrients. 2023 Oct 16;15(20):4373. doi: 10.3390/nu15204373.
2
Revisiting Telomere Shortening in Cancer.重新审视癌症中的端粒缩短。
Cells. 2019 Jan 31;8(2):107. doi: 10.3390/cells8020107.
3
Telomere Biology-Insights into an Intriguing Phenomenon.端粒生物学——对一种有趣现象的洞察
Cells. 2017 Jun 19;6(2):15. doi: 10.3390/cells6020015.
4
Quantitative 3D Telomeric Imaging of Buccal Cells Reveals Alzheimer's Disease-Specific Signatures.口腔细胞的定量三维端粒成像揭示了阿尔茨海默病的特异性特征。
J Alzheimers Dis. 2017;58(1):139-145. doi: 10.3233/JAD-161169.
本文引用的文献
1
Telomere G-tail Length is a Promising Biomarker Related to White Matter Lesions and Endothelial Dysfunction in Patients With Cardiovascular Risk: A Cross-sectional Study.端粒 G 尾长度是与心血管风险患者的白质病变和血管内皮功能障碍相关的有前途的生物标志物：一项横断面研究。
EBioMedicine. 2015 May 30;2(8):960-7. doi: 10.1016/j.ebiom.2015.05.025. eCollection 2015 Aug.
2
Telomeric repeat-containing RNA/G-quadruplex-forming sequences cause genome-wide alteration of gene expression in human cancer cells in vivo.含端粒重复序列的RNA/G-四链体形成序列在体内导致人类癌细胞中基因表达的全基因组改变。
Nucleic Acids Res. 2015 Feb 27;43(4):2022-32. doi: 10.1093/nar/gkv063. Epub 2015 Feb 4.
3
Telomeric g-tail length and hospitalization for cardiovascular events in hemodialysis patients.血液透析患者的端粒g尾长度与心血管事件住院情况
Clin J Am Soc Nephrol. 2014 Dec 5;9(12):2117-22. doi: 10.2215/CJN.10010913. Epub 2014 Sep 18.
4
Leucocyte telomere length and risk of cardiovascular disease: systematic review and meta-analysis.白细胞端粒长度与心血管疾病风险：系统评价和荟萃分析。
BMJ. 2014 Jul 8;349:g4227. doi: 10.1136/bmj.g4227.
5
Telomeres and lifestyle factors: roles in cellular aging.端粒与生活方式因素：在细胞衰老中的作用。
Mutat Res. 2012 Feb 1;730(1-2):85-9. doi: 10.1016/j.mrfmmm.2011.08.003. Epub 2011 Aug 22.
6
G-tail telomere HPA: simple measurement of human single-stranded telomeric overhangs.G尾端粒人肽抗体：人类单链端粒悬垂的简易测量方法。
Nat Methods. 2005 Nov;2(11):829-31. doi: 10.1038/nmeth797.
7
DNA processing is not required for ATM-mediated telomere damage response after TRF2 deletion.TRF2缺失后，ATM介导的端粒损伤反应不需要DNA加工。
Nat Cell Biol. 2005 Jul;7(7):712-8. doi: 10.1038/ncb1275. Epub 2005 Jun 19.
8
Mammalian telomeres end in a large duplex loop.哺乳动物的端粒以一个大的双链环结束。
Cell. 1999 May 14;97(4):503-14. doi: 10.1016/s0092-8674(00)80760-6.
9
Telomeric length and telomerase activity vary with age in peripheral blood cells obtained from normal individuals.从正常个体获取的外周血细胞中，端粒长度和端粒酶活性随年龄而变化。
Hum Genet. 1998 Apr;102(4):397-402. doi: 10.1007/s004390050711.
10
Essential role of mouse telomerase in highly proliferative organs.小鼠端粒酶在高增殖性器官中的重要作用。
Nature. 1998 Apr 9;392(6676):569-74. doi: 10.1038/33345.
Zotero 插件