巨细胞病毒感染后小鼠脾脏细胞白细胞介素-2产生受抑制的研究。
Studies of depressed interleukin-2 production by spleen cells from mice following infection with cytomegalovirus.
作者信息
Blackett S, Mims C A
机构信息
Department of Microbiology, United Medical and Dental School, Guy's Hospital, London, U.K.
出版信息
Arch Virol. 1988;99(1-2):1-8. doi: 10.1007/BF01311018.
PMID:2833199
Abstract
Spleen cells from mice infected with virulent and avirulent strains of murine cytomegalovirus showed depressed interleukin-2 production after ConA stimulation. Cell-mixing experiments indicated that this was due to a primary defect in non adherent cells. Spleen cells infected in vitro were also studied.
摘要
感染了强毒株和无毒株鼠巨细胞病毒的小鼠脾脏细胞，在刀豆蛋白A刺激后，白细胞介素-2的产生受到抑制。细胞混合实验表明，这是由于非贴壁细胞的原发性缺陷所致。还对体外感染的脾脏细胞进行了研究。
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