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Deletion of the Akt/mTORC1 Repressor REDD1 Prevents Visual Dysfunction in a Rodent Model of Type 1 Diabetes.
Diabetes. 2018 Jan;67(1):110-119. doi: 10.2337/db17-0728. Epub 2017 Oct 26.
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REDD1 Activates a ROS-Generating Feedback Loop in the Retina of Diabetic Mice.
Invest Ophthalmol Vis Sci. 2019 May 1;60(6):2369-2379. doi: 10.1167/iovs.19-26606.
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The stress response protein REDD1 promotes diabetes-induced oxidative stress in the retina by Keap1-independent Nrf2 degradation.
J Biol Chem. 2020 May 22;295(21):7350-7361. doi: 10.1074/jbc.RA120.013093. Epub 2020 Apr 15.
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NLRP3 Inflammasome Priming in the Retina of Diabetic Mice Requires REDD1-Dependent Activation of GSK3β.
Invest Ophthalmol Vis Sci. 2024 Mar 5;65(3):34. doi: 10.1167/iovs.65.3.34.
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REDD1-dependent GSK3β dephosphorylation promotes NF-κB activation and macrophage infiltration in the retina of diabetic mice.
J Biol Chem. 2023 Aug;299(8):104991. doi: 10.1016/j.jbc.2023.104991. Epub 2023 Jun 29.
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Myostatin induces DNA damage in skeletal muscle of streptozotocin-induced type 1 diabetic mice.
J Biol Chem. 2014 Feb 28;289(9):5784-98. doi: 10.1074/jbc.M113.483115. Epub 2014 Jan 14.
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Stress response protein REDD1 promotes diabetes-induced retinal inflammation by sustaining canonical NF-κB signaling.
J Biol Chem. 2022 Dec;298(12):102638. doi: 10.1016/j.jbc.2022.102638. Epub 2022 Oct 26.

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Deletion of the stress response protein REDD1 prevents sodium iodate-induced RPE damage and photoreceptor loss.
Geroscience. 2025 Apr;47(2):1789-1803. doi: 10.1007/s11357-024-01362-2. Epub 2024 Oct 5.
5
Visual Function Measurements in Eyes With Diabetic Retinopathy: An Expert Opinion on Available Measures.
Ophthalmol Sci. 2024 Apr 6;4(5):100519. doi: 10.1016/j.xops.2024.100519. eCollection 2024 Sep-Oct.
6
Sirt6 protects retinal ganglion cells and optic nerve from degeneration during aging and glaucoma.
Mol Ther. 2024 Jun 5;32(6):1760-1778. doi: 10.1016/j.ymthe.2024.04.030. Epub 2024 Apr 24.
8
NLRP3 Inflammasome Priming in the Retina of Diabetic Mice Requires REDD1-Dependent Activation of GSK3β.
Invest Ophthalmol Vis Sci. 2024 Mar 5;65(3):34. doi: 10.1167/iovs.65.3.34.
9
The stress-responsive protein REDD1 and its pathophysiological functions.
Exp Mol Med. 2023 Sep;55(9):1933-1944. doi: 10.1038/s12276-023-01056-3. Epub 2023 Sep 1.
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Visual Dysfunction in Diabetes.
Annu Rev Vis Sci. 2023 Sep 15;9:91-109. doi: 10.1146/annurev-vision-111022-123810. Epub 2023 May 10.

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The neurovascular unit and the pathophysiologic basis of diabetic retinopathy.
Graefes Arch Clin Exp Ophthalmol. 2017 Jan;255(1):1-6. doi: 10.1007/s00417-016-3548-y. Epub 2016 Nov 10.
2
The Translational Repressor 4E-BP1 Contributes to Diabetes-Induced Visual Dysfunction.
Invest Ophthalmol Vis Sci. 2016 Mar;57(3):1327-37. doi: 10.1167/iovs.15-18719.
5
REDD1 enhances protein phosphatase 2A-mediated dephosphorylation of Akt to repress mTORC1 signaling.
Sci Signal. 2014 Jul 22;7(335):ra68. doi: 10.1126/scisignal.2005103.
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Dose-ranging evaluation of intravitreal siRNA PF-04523655 for diabetic macular edema (the DEGAS study).
Invest Ophthalmol Vis Sci. 2012 Nov 15;53(12):7666-74. doi: 10.1167/iovs.12-9961.
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Post-translational regulation of FOXO.
Acta Biochim Biophys Sin (Shanghai). 2012 Nov;44(11):897-901. doi: 10.1093/abbs/gms067. Epub 2012 Aug 30.
10
Evaluation of the siRNA PF-04523655 versus ranibizumab for the treatment of neovascular age-related macular degeneration (MONET Study).
Ophthalmology. 2012 Sep;119(9):1867-73. doi: 10.1016/j.ophtha.2012.03.043. Epub 2012 Jun 8.

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