Maynard F M
Arch Phys Med Rehabil. 1986 Jan;67(1):41-4.
Based on the author's experience with more than 20 cases of immobilization hypercalcemia following spinal cord injury, current concepts of this condition are presented. Symptoms may be mild or severe: laboratory findings are essential for differential diagnosis in older individuals, in whom preinjury Paget's disease and mild primary hyperparathyroidism must be ruled out. Most cases of immobilization hypercalcemia are seen in adolescent boys following recent spinal cord injury. Besides sex (male), risk factors include age (less than 21 years), complete neurologic injuries, high cervical levels of spinal cord injury, dehydration, and a prolonged period of immobilization. A preinjury history of large ingestion of milk and/or extreme exposure to sunshine may also be contributory factors. Therapy includes vigorous hydration, saline infusions and diuretics, calcitonin, and steroids. The clinical course, without treatment, may be prolonged to 14 months, but the condition is always self-limiting.
基于作者对20多例脊髓损伤后制动性高钙血症病例的经验，本文介绍了这种病症的当前概念。症状可能轻微或严重：实验室检查结果对于老年个体的鉴别诊断至关重要，因为必须排除损伤前的佩吉特病和轻度原发性甲状旁腺功能亢进。大多数制动性高钙血症病例见于近期脊髓损伤后的青少年男性。除性别（男性）外，危险因素还包括年龄（小于21岁）、完全性神经损伤、脊髓损伤的高颈段水平、脱水以及长期制动。损伤前大量摄入牛奶和/或过度暴露于阳光下的病史也可能是促成因素。治疗包括积极补液、输注生理盐水和使用利尿剂、降钙素和类固醇。未经治疗的临床病程可能延长至14个月，但该病症总是自限性的。
