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Mmip1: a novel leucine zipper protein that reverses the suppressive effects of Mad family members on c-myc.
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Myc and Mad bHLHZ domains possess identical DNA-binding specificities but only partially overlapping functions in vivo.
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Differential effects by Mad and Max on transformation by cellular and viral oncoproteins.
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The Myc/Max/Mad network and the transcriptional control of cell behavior.
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Transcriptional control: calling in histone deacetylase.
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Inhibition of cIAP1 as a strategy for targeting c-MYC-driven oncogenic activity.
Proc Natl Acad Sci U S A. 2018 Oct 2;115(40):E9317-E9324. doi: 10.1073/pnas.1807711115. Epub 2018 Sep 4.
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The MYC transcription factor network: balancing metabolism, proliferation and oncogenesis.
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Targeting oncogenic Myc as a strategy for cancer treatment.
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MYC degradation.
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Inhibitor of apoptosis (IAP) proteins-modulators of cell death and inflammation.
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Transcriptional amplification in tumor cells with elevated c-Myc.
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Detection of sequential polyubiquitylation on a millisecond timescale.
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Rapid E2-E3 assembly and disassembly enable processive ubiquitylation of cullin-RING ubiquitin ligase substrates.
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The suppression of MAD1 by AKT-mediated phosphorylation activates MAD1 target genes transcription.
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Reflecting on 25 years with MYC.
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