Dysregulation of lung myeloid cells in COVID-19.
作者信息
Salomé Bérengère, Magen Assaf
机构信息
Sinai Immunology Review Project, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
出版信息
Nat Rev Immunol. 2020 May;20(5):277. doi: 10.1038/s41577-020-0303-8.
Abstract
摘要
相似文献
1
Dysregulation of lung myeloid cells in COVID-19.新型冠状病毒肺炎中肺髓样细胞的失调
Nat Rev Immunol. 2020 May;20(5):277. doi: 10.1038/s41577-020-0303-8.
2
Pathological features of COVID-19-associated lung injury: a preliminary proteomics report based on clinical samples.基于临床样本的 COVID-19 相关肺损伤的病理特征：初步蛋白质组学报告。
Signal Transduct Target Ther. 2020 Oct 15;5(1):240. doi: 10.1038/s41392-020-00355-9.
3
Immunothrombotic Dysregulation in COVID-19 Pneumonia Is Associated With Respiratory Failure and Coagulopathy.新型冠状病毒肺炎肺炎中的免疫血栓调节紊乱与呼吸衰竭和凝血病有关。
Circulation. 2020 Sep 22;142(12):1176-1189. doi: 10.1161/CIRCULATIONAHA.120.048488. Epub 2020 Jul 28.
4
Complex Immune Dysregulation in COVID-19 Patients with Severe Respiratory Failure.COVID-19 患者严重呼吸衰竭的复杂免疫失调。
Cell Host Microbe. 2020 Jun 10;27(6):992-1000.e3. doi: 10.1016/j.chom.2020.04.009. Epub 2020 Apr 21.
5
Tobacco, but Not Nicotine and Flavor-Less Electronic Cigarettes, Induces ACE2 and Immune Dysregulation.烟草，而非尼古丁和无味道电子烟，会诱导 ACE2 和免疫失调。
Int J Mol Sci. 2020 Jul 31;21(15):5513. doi: 10.3390/ijms21155513.
6
Computing the Effects of SARS-CoV-2 on Respiration Regulatory Mechanisms in COVID-19.计算 SARS-CoV-2 对 COVID-19 中呼吸调节机制的影响。
ACS Chem Neurosci. 2020 Aug 19;11(16):2416-2421. doi: 10.1021/acschemneuro.0c00349. Epub 2020 Aug 11.
7
In silico immune infiltration profiling combined with functional enrichment analysis reveals a potential role for naïve B cells as a trigger for severe immune responses in the lungs of COVID-19 patients.基于计算的免疫浸润分析结合功能富集分析揭示了幼稚 B 细胞在 COVID-19 患者肺部引发严重免疫反应的潜在作用。
PLoS One. 2020 Dec 2;15(12):e0242900. doi: 10.1371/journal.pone.0242900. eCollection 2020.
8
COVID-19: Catastrophic Cause of Acute Lung Injury.新型冠状病毒肺炎：急性肺损伤的灾难性病因
S D Med. 2020 Jun;73(6):252-260.
9
Myeloid tissue factor does not modulate lung inflammation or permeability during experimental acute lung injury.在实验性急性肺损伤期间，髓系组织因子不会调节肺部炎症或通透性。
Sci Rep. 2016 Feb 29;6:22249. doi: 10.1038/srep22249.
10
General, but not myeloid or type II lung epithelial cell, myeloid differentiation factor 88 deficiency abrogates house dust mite induced allergic lung inflammation.一般而言，但髓系细胞或II型肺上皮细胞除外，髓系分化因子88缺乏可消除屋尘螨诱导的过敏性肺部炎症。
Clin Exp Immunol. 2017 Feb;187(2):204-212. doi: 10.1111/cei.12867. Epub 2016 Oct 13.
引用本文的文献
1
Differences in clinical characteristics and liver injury between patients diagnosed with the Omicron subvariant BA.5.2 and the prototype of SARS-CoV-2: a single center retrospective study.奥密克戎变异株 BA.5.2 与 SARS-CoV-2 原型株所致临床特征及肝损伤的差异：一项单中心回顾性研究。
BMC Gastroenterol. 2023 Aug 8;23(1):271. doi: 10.1186/s12876-023-02907-z.
2
Potential plants for inflammatory dysfunction in the SARS-CoV-2 infection.新型冠状病毒感染中潜在的炎症功能障碍植物。
Inflammopharmacology. 2022 Jun;30(3):749-773. doi: 10.1007/s10787-022-00981-5. Epub 2022 Apr 7.
3
Humoral immune mechanisms involved in protective and pathological immunity during COVID-19.体液免疫机制在 COVID-19 保护性和病理性免疫中的作用。
Hum Immunol. 2021 Oct;82(10):733-745. doi: 10.1016/j.humimm.2021.06.011. Epub 2021 Jul 1.
4
Differences in clinical characteristics and liver injury between suspected and confirmed COVID-19 patients in Jingzhou, Hubei Province of China.中国湖北省荆州市疑似和确诊 COVID-19 患者的临床特征和肝损伤差异。
Medicine (Baltimore). 2021 May 14;100(19):e25913. doi: 10.1097/MD.0000000000025913.
5
Risk factors for adverse outcomes of COVID-19 patients: Possible basis for diverse responses to the novel coronavirus SARS-CoV-2.COVID-19 患者不良结局的风险因素：对新型冠状病毒 SARS-CoV-2 不同反应的可能基础。
Life Sci. 2021 Jul 15;277:119503. doi: 10.1016/j.lfs.2021.119503. Epub 2021 Apr 15.
6
Nanocarrier vaccines for SARS-CoV-2.用于 SARS-CoV-2 的纳米载体疫苗。
Adv Drug Deliv Rev. 2021 Apr;171:215-239. doi: 10.1016/j.addr.2021.01.002. Epub 2021 Jan 9.
7
Preventing the development of severe COVID-19 by modifying immunothrombosis.通过调节免疫血栓来预防重症 COVID-19 的发生。
Life Sci. 2021 Jan 1;264:118617. doi: 10.1016/j.lfs.2020.118617. Epub 2020 Oct 20.
8
Can endolysosomal deacidification and inhibition of autophagy prevent severe COVID-19?内溶酶体去酸化和自噬抑制能否预防重症 COVID-19？
Life Sci. 2020 Dec 1;262:118541. doi: 10.1016/j.lfs.2020.118541. Epub 2020 Oct 6.
9
Obesity, COVID-19 and innate immunometabolism.肥胖、COVID-19 和先天免疫代谢。
Br J Nutr. 2021 Mar 28;125(6):628-632. doi: 10.1017/S0007114520003529. Epub 2020 Sep 7.
10
The angiocrine Rspondin3 instructs interstitial macrophage transition via metabolic-epigenetic reprogramming and resolves inflammatory injury.血管分泌因子 Rspondin3 通过代谢-表观遗传重编程来指导间充质巨噬细胞的转化，并解决炎症损伤。
Nat Immunol. 2020 Nov;21(11):1430-1443. doi: 10.1038/s41590-020-0764-8. Epub 2020 Aug 24.
Zotero 插件