感染毒力梅毒螺旋体的家兔巨噬细胞移动抑制的发展
Development of of macrophage migration inhibition in rabbits infected with virulent Treponema pallidum.
作者信息
Pavia C S, Folds J D, Baseman J B
出版信息
Infect Immun. 1977 Sep;17(3):651-4. doi: 10.1128/iai.17.3.651-654.1977.
Abstract
Peritoneal exudate cells from rabbits infected with Treponema pallidum Nichols were used as indicators of macrophage migration inhibitory factor activity. Between 3 and 15 weeks after infection, the migration of peritoneal exudate cells was inhibited in the presence of 3 to 25 microgram of T. phagedenis biovar Reiter protein per ml. Before this period, the migration patterns of peritoneal exudate cells from infected animals were uninhibited and similar to those from noninfected control rabbits. These observations were correlated with the development of active cell-mediated immunity during experimental T. pallidum infection.
摘要
将感染梅毒螺旋体Nichols株的家兔的腹腔渗出细胞用作巨噬细胞移动抑制因子活性的指标。感染后3至15周期间，每毫升存在3至25微克蚀疮性溃疡螺旋体生物变种Reiter蛋白时，腹腔渗出细胞的移动受到抑制。在此之前，感染动物的腹腔渗出细胞的移动模式未受抑制，且与未感染的对照家兔的移动模式相似。这些观察结果与实验性梅毒螺旋体感染期间活性细胞介导免疫的发展相关。
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