Scott J A
National Radiological Protection Board, Chilton, Didcot, Oxon, UK.
Phys Med Biol. 1988 Feb;33(2):243-57. doi: 10.1088/0031-9155/33/2/004.
Long-term industrial exposure to low levels of infrared radiation has for many years been associated with the development of cataracts; the injury mechanism is widely held to be thermal. A finite element model of the human eye is employed to calculate the temperature rises experienced by the intraocular media when exposed to infrared radiation. The model is used to calculate transient and steady-state temperature distributions for various exposure times and a range of incident irradiances. The effect of the eye's natural cooling mechanisms on the heating is investigated. Specific absorption rates in the infrared irradiated eye are presented. For a radiation source of 1500 degrees C, absorption of radiant energy by the iris and the lens combined with conduction of heat from the anterior regions is found to be responsible for increases in the lens temperature of 1-2 degrees C, but under extreme exposure conditions the temperature rises are found to be substantially higher.
长期在工业环境中接触低水平红外辐射多年来一直被认为与白内障的形成有关；损伤机制普遍被认为是热效应。采用人眼的有限元模型来计算眼内介质在暴露于红外辐射时所经历的温度升高。该模型用于计算不同暴露时间和一系列入射辐照度下的瞬态和稳态温度分布。研究了眼睛自然冷却机制对加热的影响。给出了红外辐照眼睛中的比吸收率。对于1500摄氏度的辐射源，发现虹膜和晶状体对辐射能的吸收以及来自前部区域的热传导导致晶状体温度升高1 - 2摄氏度，但在极端暴露条件下，温度升高要高得多。
