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The role of MHC class II antigens in the flare up reaction of antigen-induced arthritis in mice.

作者信息

van den Broek M F, van den Berg W B, van de Putte L B

出版信息

Agents Actions. 1986 Dec;19(5-6):328-30. doi: 10.1007/BF01971241.

DOI:10.1007/BF01971241
PMID:3469892
Abstract
摘要

相似文献

1
The role of MHC class II antigens in the flare up reaction of antigen-induced arthritis in mice.MHC II类抗原在小鼠抗原诱导性关节炎复发反应中的作用。
Agents Actions. 1986 Dec;19(5-6):328-30. doi: 10.1007/BF01971241.
2
Monoclonal anti-Ia antibodies suppress the flare up reaction of antigen induced arthritis in mice.单克隆抗Ia抗体可抑制小鼠抗原诱导性关节炎的突发反应。
Clin Exp Immunol. 1986 Nov;66(2):320-30.
3
Collagen type II (CII)-specific antibodies induce arthritis in the absence of T or B cells but the arthritis progression is enhanced by CII-reactive T cells.II型胶原（CII）特异性抗体在缺乏T细胞或B细胞的情况下可诱发关节炎，但CII反应性T细胞会加速关节炎的进展。
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4
Autoimmunity and inflammation are independent of class II transactivator type PIV-dependent class II major histocompatibility complex expression in peripheral tissues during collagen-induced arthritis.在胶原诱导的关节炎中，自身免疫和炎症与外周组织中II类反式激活因子PIV依赖性II类主要组织相容性复合体的表达无关。
Arthritis Rheum. 2011 Nov;63(11):3354-63. doi: 10.1002/art.30522.
5
Effects of C2ta genetic polymorphisms on MHC class II expression and autoimmune diseases.C2ta基因多态性对MHC II类分子表达及自身免疫性疾病的影响。
Immunology. 2017 Apr;150(4):408-417. doi: 10.1111/imm.12692. Epub 2016 Dec 22.
6
Type II collagen-induced arthritis in mice. I. Major histocompatibility complex (I region) linkage and antibody correlates.小鼠Ⅱ型胶原诱导性关节炎。I. 主要组织相容性复合体（I区）连锁及抗体相关性
J Exp Med. 1981 Sep 1;154(3):688-700. doi: 10.1084/jem.154.3.688.
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In vivo modulation of murine collagen induced arthritis.小鼠胶原诱导性关节炎的体内调节
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8
Expression of a transgenic class II Ab gene confers susceptibility to collagen-induced arthritis.转基因II类抗体基因的表达赋予对胶原诱导性关节炎的易感性。
Eur J Immunol. 1994 Jul;24(7):1698-702. doi: 10.1002/eji.1830240736.
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Major histocompatibility complex-restricted and unrestricted T helper cells recognizing minor histocompatibility antigens of B cell surfaces.识别B细胞表面次要组织相容性抗原的主要组织相容性复合体限制和非限制的辅助性T细胞。
Eur J Immunol. 1980 Jul;10(7):535-41. doi: 10.1002/eji.1830100710.
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Flare-up of antigen-induced arthritis in mice after challenge with intravenous antigen: studies on the characteristics of and mechanisms involved in the reaction.静脉注射抗原激发后小鼠抗原诱导性关节炎的复发：关于该反应特征及相关机制的研究
Clin Exp Immunol. 1984 Feb;55(2):287-94.

引用本文的文献

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Streptococcal cell wall-induced arthritis and flare-up reaction in mice induced by homologous or heterologous cell walls.链球菌细胞壁诱导的关节炎以及同源或异源细胞壁在小鼠中引发的炎症反应。
Am J Pathol. 1988 Oct;133(1):139-49.

本文引用的文献

1
Disappearance and reappearance of B cells after in vivo treatment with monoclonal anti-I-A antibodies.用单克隆抗I-A抗体进行体内治疗后B细胞的消失与再现。
Proc Natl Acad Sci U S A. 1984 May;81(9):2855-8. doi: 10.1073/pnas.81.9.2855.
2
Treatment of (NZB x NZW)F1 disease with anti-I-A monoclonal antibodies.用抗I-A单克隆抗体治疗（新西兰黑鼠×新西兰白鼠）F1代疾病。
J Exp Med. 1983 Oct 1;158(4):1350-5. doi: 10.1084/jem.158.4.1350.
3
Inhibitory activity of antibodies to human Ia-like determinants: comparison of intact and pepsin-digested antibodies.
抗人Ia样决定簇抗体的抑制活性：完整抗体与胃蛋白酶消化抗体的比较
J Immunol. 1983 Aug;131(2):725-30.
4
In vivo therapy with monoclonal anti-I-A antibody suppresses immune responses to acetylcholine receptor.用单克隆抗I-A抗体进行体内治疗可抑制对乙酰胆碱受体的免疫反应。
Proc Natl Acad Sci U S A. 1983 May;80(9):2713-7. doi: 10.1073/pnas.80.9.2713.
5
Flare-up of antigen-induced arthritis in mice after challenge with intravenous antigen: effects of pre-treatment with cobra venom factor and anti-lymphocyte serum.静脉注射抗原攻击后小鼠抗原诱导性关节炎的复发：眼镜蛇毒因子和抗淋巴细胞血清预处理的影响
Clin Exp Immunol. 1984 Sep;57(3):520-8.
6
Conversion of immunity to suppression by in vivo administration of I-A subregion-specific antibodies.通过体内给予I-A亚区特异性抗体将免疫转化为抑制。
J Exp Med. 1982 Aug 1;156(2):480-91. doi: 10.1084/jem.156.2.480.
7
Flare-up of antigen-induced arthritis in mice after challenge with intravenous antigen: studies on the characteristics of and mechanisms involved in the reaction.静脉注射抗原激发后小鼠抗原诱导性关节炎的复发：关于该反应特征及相关机制的研究
Clin Exp Immunol. 1984 Feb;55(2):287-94.
8
Anti I-A antibody suppresses active encephalomyelitis: treatment model for diseases linked to IR genes.抗I-A抗体可抑制活动性脑脊髓炎：与免疫反应基因相关疾病的治疗模型。
J Exp Med. 1983 Oct 1;158(4):1362-7. doi: 10.1084/jem.158.4.1362.
9
Antigen-specific human T lymphocyte clones: mechanisms of inhibition of proliferative responses by xenoantiserum to human nonpolymorphic HLA-DR antigens.
J Immunol. 1982 Sep;129(3):1085-90.
10
Type II collagen-induced arthritis in mice. III. Suppression of arthritis by using monoclonal and polyclonal anti-Ia antisera.
J Immunol. 1985 Apr;134(4):2366-74.