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A subpopulation of t(2;14)(p11;q32) cells in ataxia telangiectasia B lymphocytes.

作者信息

Butterworth S V, Taylor A M

出版信息

Hum Genet. 1986 Aug;73(4):346-9. doi: 10.1007/BF00279098.

DOI:10.1007/BF00279098
PMID:3488948
Abstract

Partially purified B cells from ataxia telangiectasia (A-T) patients and normal individuals were stimulated with Staphylococcus aureus Cowan I organisms (SAC). High levels of apparently random rearrangements were seen in the A-T B cells only. In addition a t(2;14)(p11;q32) rearrangement was identified in B cells from more than one patient.

摘要

相似文献

1
A subpopulation of t(2;14)(p11;q32) cells in ataxia telangiectasia B lymphocytes.
Hum Genet. 1986 Aug;73(4):346-9. doi: 10.1007/BF00279098.
2
Characterization of a B-lymphocyte t(2;14) (p11;q32) translocation from an ataxia telangiectasia patient conferring a proliferative advantage on cells in vitro.一名共济失调毛细血管扩张症患者的B淋巴细胞t(2;14)(p11;q32)易位的特征，该易位赋予细胞体外增殖优势。
Cytogenet Cell Genet. 1991;56(2):91-8. doi: 10.1159/000133057.
3
New data on clonal anomalies of chromosome 14 in ataxia telangiectasia: tct(14;14) and inv(14).共济失调毛细血管扩张症中14号染色体克隆性异常的新数据：tct(14;14)和inv(14)
Hum Genet. 1986 Jan;72(1):22-4. doi: 10.1007/BF00278811.
4
Probable involvement of immunoglobulin superfamily genes in most recurrent chromosomal rearrangements from ataxia telangiectasia.
Hum Genet. 1986 Mar;72(3):210-4. doi: 10.1007/BF00291879.
5
Tandem translocation t(14;14) in isolated and clonal cells in ataxia telangiectasia are different.共济失调毛细血管扩张症中分离的克隆细胞中的串联易位t(14;14)有所不同。
Hum Genet. 1983;63(4):320-2. doi: 10.1007/BF00274754.
6
The chromosome breakpoint at 14q32 in an ataxia telangiectasia t(14;14) T cell clone is different from the 14q32 breakpoint in Burkitts and an inv(14) T cell lymphoma.共济失调毛细血管扩张症t(14;14) T细胞克隆中14q32处的染色体断点与伯基特淋巴瘤及inv(14) T细胞淋巴瘤中14q32处的断点不同。
Hum Genet. 1986 Jul;73(3):254-9. doi: 10.1007/BF00401239.
7
High frequencies of inversions and translocations of chromosomes 7 and 14 in ataxia telangiectasia.共济失调毛细血管扩张症中7号和14号染色体的倒位和易位频率较高。
Mutat Res. 1980 Feb;69(2):369-74. doi: 10.1016/0027-5107(80)90101-3.
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Neoplasia and chromosomal breakage in ataxia-telangiectasia: a 2:14 translocation.共济失调毛细血管扩张症中的肿瘤形成与染色体断裂：一种2:14易位
Kroc Found Ser. 1985;19:197-203.
9
VP16 hypersensitivity and increased faulty recombination in ataxia telangiectasia lymphocytes characterized by the tandem translocation t(14;14)(q11;q32).以串联易位t(14;14)(q11;q32)为特征的共济失调毛细血管扩张症淋巴细胞中，VP16超敏反应及错误重组增加。
Carcinogenesis. 1996 Feb;17(2):203-7. doi: 10.1093/carcin/17.2.203.
10
Evolution of chromosomal abnormalities in sequential cytogenetic studies of ataxia telangiectasia.共济失调毛细血管扩张症连续细胞遗传学研究中染色体异常的演变
Hum Genet. 1980;55(1):23-9. doi: 10.1007/BF00329122.

引用本文的文献

1
Growth of large chromosomally abnormal T cell clones in ataxia telangiectasia patients is associated with translocation at 14q11. A model for other T cell neoplasia.共济失调毛细血管扩张症患者中大型染色体异常T细胞克隆的生长与14q11处的易位有关。这是其他T细胞肿瘤形成的一种模式。
Hum Genet. 1987 Aug;76(4):389-95. doi: 10.1007/BF00272451.
2
Analysis of 7 polymorphic markers at chromosome 11q22-23 in 35 ataxia telangiectasia families; further evidence of linkage.对35个共济失调毛细血管扩张症家系中11号染色体q22 - 23区域的7个多态性标记进行分析；连锁的进一步证据。
Hum Genet. 1990 Jul;85(2):215-20. doi: 10.1007/BF00193199.

本文引用的文献

1
Spontaneous cytogenetic abnormalities in lymphocytes from thirteen patients with ataxia telangiectasia.13例共济失调毛细血管扩张症患者淋巴细胞的自发细胞遗传学异常。
Int J Cancer. 1981 Mar 15;27(3):311-9. doi: 10.1002/ijc.2910270309.
2
Tandem translocation t(14;14) in isolated and clonal cells in ataxia telangiectasia are different.共济失调毛细血管扩张症中分离的克隆细胞中的串联易位t(14;14)有所不同。
Hum Genet. 1983;63(4):320-2. doi: 10.1007/BF00274754.
3
Inversion of chromosome 14 marks human T-cell chronic lymphocytic leukaemia.14号染色体倒位标志着人类T细胞慢性淋巴细胞白血病。
Nature. 1984;308(5962):858-60. doi: 10.1038/308858a0.
4
Cytogenetic studies on patients with chronic T cell leukemia/lymphoma.慢性T细胞白血病/淋巴瘤患者的细胞遗传学研究。
Blood. 1984 May;63(5):1028-38.
5
Common region on chromosome 14 in T-cell leukemia and lymphoma.T细胞白血病和淋巴瘤中14号染色体上的共同区域。
Science. 1984 Dec 21;226(4681):1445-7. doi: 10.1126/science.6438800.
6
Chromosomal aberrations in a case of T-cell CLL with concomitant IgA myeloma.
Int J Cancer. 1983 Oct 15;32(4):431-5. doi: 10.1002/ijc.2910320406.
7
Chromosomal translocation involving the immunoglobulin kappa-chain and heavy-chain loci in a child with chronic lymphocytic leukemia.
N Engl J Med. 1983 Sep 8;309(10):590-4. doi: 10.1056/NEJM198309083091005.
8
Localization of the human alpha-globin gene cluster to the short arm of chromosome 16 (16p12-16pter) by hybridization in situ.
J Mol Biol. 1982 Apr 5;156(2):269-78. doi: 10.1016/0022-2836(82)90328-x.
9
Inversion (14)(q12qter) or (q11.2q32.3): the most frequently acquired rearrangement in lymphocytes.倒位（14）（q12q末端）或（q11.2q32.3）：淋巴细胞中最常见的获得性重排。
Hum Genet. 1985;71(1):19-21. doi: 10.1007/BF00295660.
10
T-cell receptor genes and ataxia telangiectasia.T细胞受体基因与共济失调毛细血管扩张症
Nature. 1985;313(5999):186. doi: 10.1038/313186a0.