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1
T-lymphocyte involvement in abscess formation in nonimmune mice.T淋巴细胞参与非免疫小鼠脓肿的形成。
Infect Immun. 1986 May;52(2):633-6. doi: 10.1128/iai.52.2.633-636.1986.
2
Evidence for T cell-dependent immunity to Bacteroides fragilis in an intraabdominal abscess model.腹腔脓肿模型中针对脆弱拟杆菌的T细胞依赖性免疫的证据。
J Clin Invest. 1982 Jan;69(1):9-16. doi: 10.1172/jci110445.
3
Cellular control of abscess formation: role of T cells in the regulation of abscesses formed in response to Bacteroides fragilis.脓肿形成的细胞调控:T细胞在脆弱拟杆菌诱导形成的脓肿调节中的作用
J Immunol. 1986 Jul 1;137(1):341-6.
4
Use of a model of intraabdominal sepsis for studies of the pathogenicity of Bacteroides fragilis.使用腹腔内脓毒症模型研究脆弱拟杆菌的致病性。
Rev Infect Dis. 1984 Mar-Apr;6 Suppl 1:S91-5. doi: 10.1093/clinids/6.supplement_1.s91.
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Neutrophil activity in abscess-bearing mice: comparative studies with neutrophils isolated from peripheral blood, elicited peritoneal exudates, and abscesses.有脓肿小鼠的中性粒细胞活性:与从外周血、诱导产生的腹腔渗出液和脓肿中分离出的中性粒细胞的对比研究。
Infect Immun. 1986 Mar;51(3):936-41. doi: 10.1128/iai.51.3.936-941.1986.
6
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Immunization-induced changes in later intra-abdominal abscess formation are mediated by cellular and humoral mechanisms.免疫诱导的后期腹腔内脓肿形成变化是由细胞和体液机制介导的。
Curr Surg. 1990 Sep-Oct;47(5):331-4.
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T cell regulation of Bacteroides fragilis-induced intraabdominal abscesses.T细胞对脆弱拟杆菌诱导的腹腔内脓肿的调节作用。
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CD4+ T cells mediate abscess formation in intra-abdominal sepsis by an IL-17-dependent mechanism.CD4+ T细胞通过白细胞介素-17依赖机制介导腹腔内脓毒症中的脓肿形成。
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Role of T lymphocytes in liver abscess formation by Bacteroides fragilis in mice.脆弱拟杆菌致小鼠肝脓肿形成中 T 淋巴细胞的作用。
Infect Immun. 2011 Jun;79(6):2234-40. doi: 10.1128/IAI.01167-10. Epub 2011 Mar 28.

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Differences in host susceptibility to disease progression in the human challenge model of Haemophilus ducreyi infection.在杜克雷嗜血杆菌感染的人体激发模型中宿主对疾病进展易感性的差异。
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2
Bacterial pathogens induce abscess formation by CD4(+) T-cell activation via the CD28-B7-2 costimulatory pathway.细菌病原体通过CD28 - B7 - 2共刺激途径激活CD4(+) T细胞,从而诱导脓肿形成。
Infect Immun. 2000 Dec;68(12):6650-5. doi: 10.1128/IAI.68.12.6650-6655.2000.
3
Use of lymphokines in treatment of experimental intra-abdominal abscess caused by Bacteroides fragilis.淋巴因子在治疗脆弱拟杆菌引起的实验性腹腔内脓肿中的应用。
Infect Immun. 1988 Sep;56(9):2369-72. doi: 10.1128/iai.56.9.2369-2372.1988.
4
Experimental Porphyromonas gingivalis infection in nonimmune athymic BALB/c mice.非免疫性无胸腺BALB/c小鼠的牙龈卟啉单胞菌实验性感染
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5
Retrovirus-induced immunodeficiency in mice exacerbates gastrointestinal candidiasis.逆转录病毒诱导的小鼠免疫缺陷会加剧胃肠道念珠菌病。
Infect Immun. 1992 Oct;60(10):4168-78. doi: 10.1128/iai.60.10.4168-4178.1992.

本文引用的文献

1
Host factors in the formation of abscesses.脓肿形成中的宿主因素。
J Infect Dis. 1980 Jul;142(1):40-9. doi: 10.1093/infdis/142.1.40.
2
A quantitative model for subcutaneous abscess formation in mice.小鼠皮下脓肿形成的定量模型。
Br J Exp Pathol. 1980 Feb;61(1):97-107.
3
Evidence for T cell-dependent immunity to Bacteroides fragilis in an intraabdominal abscess model.腹腔脓肿模型中针对脆弱拟杆菌的T细胞依赖性免疫的证据。
J Clin Invest. 1982 Jan;69(1):9-16. doi: 10.1172/jci110445.
4
The role of cell-mediated immunity in bacterial infections.细胞介导的免疫在细菌感染中的作用。
Rev Infect Dis. 1981 Nov-Dec;3(6):1221-50. doi: 10.1093/clinids/3.6.1221.
5
Intra-abdominal abscess formation in mice: quantitative studies on bacteria and abscess-potentiating agents.小鼠腹腔内脓肿形成:关于细菌和脓肿促发剂的定量研究
Br J Exp Pathol. 1983 Aug;64(4):345-53.
6
Cellular immunity to Bacteroides fragilis capsular polysaccharide.对脆弱拟杆菌荚膜多糖的细胞免疫
J Exp Med. 1982 Apr 1;155(4):1188-97. doi: 10.1084/jem.155.4.1188.
7
Role of cell surface receptors in the regulation of intracellular killing of bacteria by murine peritoneal exudate neutrophils.细胞表面受体在小鼠腹膜渗出中性粒细胞对细菌的细胞内杀伤调节中的作用。
Infect Immun. 1986 Apr;52(1):245-51. doi: 10.1128/iai.52.1.245-251.1986.
8
Neutrophil activity in abscess-bearing mice: comparative studies with neutrophils isolated from peripheral blood, elicited peritoneal exudates, and abscesses.有脓肿小鼠的中性粒细胞活性:与从外周血、诱导产生的腹腔渗出液和脓肿中分离出的中性粒细胞的对比研究。
Infect Immun. 1986 Mar;51(3):936-41. doi: 10.1128/iai.51.3.936-941.1986.
9
A soluble suppressor T cell factor protects against experimental intraabdominal abscesses.一种可溶性抑制性T细胞因子可预防实验性腹腔脓肿。
J Clin Invest. 1985 Mar;75(3):1023-7. doi: 10.1172/JCI111763.
10
Response of congenitally athymic (nude) mice to infection with Mycobacterium bovis (strain BCG).先天性无胸腺(裸)小鼠对牛分枝杆菌(卡介苗菌株)感染的反应。
J Natl Cancer Inst. 1975 Jun;54(6):1419-26. doi: 10.1093/jnci/54.6.1419.

T淋巴细胞参与非免疫小鼠脓肿的形成。

T-lymphocyte involvement in abscess formation in nonimmune mice.

作者信息

Nulsen M F, Finlay-Jones J J, McDonald P J

出版信息

Infect Immun. 1986 May;52(2):633-6. doi: 10.1128/iai.52.2.633-636.1986.

DOI:10.1128/iai.52.2.633-636.1986
PMID:3516885
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC261051/
Abstract

Athymic mice formed significantly smaller abscesses than euthymic mice in response to the intraperitoneal inoculation of an abscess-inducing mixture of Escherichia coli, Bacteroides fragilis, and autoclaved colonic contents, an abscess-potentiating agent. Adoptive transfer of nonimmune, Thy-1-positive spleen cells to athymic mice restored their ability to make abscesses of sizes similar to those in controls, indicating that T lymphocytes contribute to abscess formation in normal mice.

摘要

在腹腔接种由大肠杆菌、脆弱拟杆菌和经高压灭菌的结肠内容物(一种脓肿增强剂)组成的脓肿诱导混合物后,无胸腺小鼠形成的脓肿明显小于有胸腺小鼠。将非免疫的、Thy-1阳性的脾细胞过继转移到无胸腺小鼠中,恢复了它们形成与对照组大小相似脓肿的能力,这表明T淋巴细胞在正常小鼠的脓肿形成中起作用。