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T-lymphocyte involvement in abscess formation in nonimmune mice.
Infect Immun. 1986 May;52(2):633-6. doi: 10.1128/iai.52.2.633-636.1986.
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Use of a model of intraabdominal sepsis for studies of the pathogenicity of Bacteroides fragilis.
Rev Infect Dis. 1984 Mar-Apr;6 Suppl 1:S91-5. doi: 10.1093/clinids/6.supplement_1.s91.
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CD4+ T cells mediate abscess formation in intra-abdominal sepsis by an IL-17-dependent mechanism.
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Role of T lymphocytes in liver abscess formation by Bacteroides fragilis in mice.
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Bacterial pathogens induce abscess formation by CD4(+) T-cell activation via the CD28-B7-2 costimulatory pathway.
Infect Immun. 2000 Dec;68(12):6650-5. doi: 10.1128/IAI.68.12.6650-6655.2000.
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Use of lymphokines in treatment of experimental intra-abdominal abscess caused by Bacteroides fragilis.
Infect Immun. 1988 Sep;56(9):2369-72. doi: 10.1128/iai.56.9.2369-2372.1988.
4
Experimental Porphyromonas gingivalis infection in nonimmune athymic BALB/c mice.
Infect Immun. 1991 Dec;59(12):4706-9. doi: 10.1128/iai.59.12.4706-4709.1991.
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Retrovirus-induced immunodeficiency in mice exacerbates gastrointestinal candidiasis.
Infect Immun. 1992 Oct;60(10):4168-78. doi: 10.1128/iai.60.10.4168-4178.1992.

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Host factors in the formation of abscesses.
J Infect Dis. 1980 Jul;142(1):40-9. doi: 10.1093/infdis/142.1.40.
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The role of cell-mediated immunity in bacterial infections.
Rev Infect Dis. 1981 Nov-Dec;3(6):1221-50. doi: 10.1093/clinids/3.6.1221.
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Cellular immunity to Bacteroides fragilis capsular polysaccharide.
J Exp Med. 1982 Apr 1;155(4):1188-97. doi: 10.1084/jem.155.4.1188.
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A soluble suppressor T cell factor protects against experimental intraabdominal abscesses.
J Clin Invest. 1985 Mar;75(3):1023-7. doi: 10.1172/JCI111763.
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Response of congenitally athymic (nude) mice to infection with Mycobacterium bovis (strain BCG).
J Natl Cancer Inst. 1975 Jun;54(6):1419-26. doi: 10.1093/jnci/54.6.1419.

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