Vargas Roth A A, Moscatelli Marco, Vaz de Lima Marcos, Ramírez León Jorge Felipe, Lorio Morgan P, Fiorelli Rossano Kepler Alvim, Telfeian Albert E, Fiallos John, Braxton Ernest, Song Michael, Lewandrowski Kai-Uwe
RIWO Spine Center of Excellence, Department of Neurosurgery, Foundation Hospital Centro Médico Campinas, Campinas 13101-627, SP, Brazil.
Clinica NeuroLife, Natal 59054-630, RN, Brazil.
J Pers Med. 2023 Feb 22;13(3):381. doi: 10.3390/jpm13030381.
: Seizures, neurological deficits, bradycardia, and, in the worst cases, cardiac arrest may occur following incidental durotomy during routine lumbar endoscopy. Therefore, we set out to measure the intraoperative epidural pressure during lumbar endoscopic decompression surgery. : We conducted a retrospective observational cohort study to obtain intraoperative epidural measurements with an epidural catheter-pressure transducer assembly through the spinal endoscope on 15 patients who underwent lumbar endoscopic decompression of symptomatic lumbar herniated discs and spinal stenosis. The endoscopic interlaminar technique was employed. : There were six (40.0%) female and nine (60.0%) male patients aged 49.0667 ± 11.31034, ranging from 36 to 72 years, with an average follow-up of 35.15 ± 12.48 months. Three of the fifteen patients had seizures with durotomy and one of these three had intracranial air on their postoperative brain CT. Another patient developed spinal headaches and diplopia on postoperative day one when her deteriorating neurological function was investigated with a brain computed tomography (CT) scan, showing an intraventricular hemorrhage consistent with a Fisher Grade IV subarachnoid hemorrhage. A CT angiogram did not show any abnormalities. Pressure recordings in the epidural space in nine patients ranged from 20 to 29 mm Hg with a mean of 24.33 mm Hg. : Most incidental durotomies encountered during lumbar interlaminar endoscopy can be managed without formal repair and supportive care measures. The intradural spread of irrigation fluid and intraoperatively used drugs and air entrapment through an unrecognized durotomy should be suspected if patients deteriorate in the recovery room. Ascending paralysis may cause nausea, vomiting, upper and lower motor neuron symptoms, cranial nerve palsies, hypotension, bradycardia, and respiratory and cardiac arrest. The recovery team should be prepared to manage these complications.
在常规腰椎内镜检查期间意外切开硬脊膜后,可能会发生癫痫发作、神经功能缺损、心动过缓,在最严重的情况下,还可能发生心脏骤停。因此,我们着手测量腰椎内镜减压手术期间的术中硬膜外压力。我们进行了一项回顾性观察队列研究,通过脊髓内窥镜使用硬膜外导管-压力传感器组件,对15例行有症状腰椎间盘突出症和腰椎管狭窄症腰椎内镜减压术的患者进行术中硬膜外测量。采用内镜下椎间孔技术。有6名(40.0%)女性和9名(60.0%)男性患者,年龄49.0667±11.31034岁,年龄范围为36至72岁,平均随访35.15±12.48个月。15名患者中有3名在切开硬脊膜后发生癫痫发作,这3名患者中有1名术后脑部CT显示颅内积气。另一名患者在术后第1天出现脊髓性头痛和复视,当时对其神经功能恶化情况进行脑部计算机断层扫描(CT)检查,显示与Fisher IV级蛛网膜下腔出血一致的脑室内出血。CT血管造影未显示任何异常。9名患者硬膜外间隙压力记录范围为20至29毫米汞柱,平均为24.33毫米汞柱。在腰椎椎间孔内镜检查期间遇到的大多数意外硬脊膜切开术无需正式修复和支持性护理措施即可处理。如果患者在恢复室病情恶化,应怀疑冲洗液、术中使用的药物通过未被识别的硬脊膜切开术发生硬膜内扩散以及空气滞留。上行性麻痹可能导致恶心、呕吐、上下运动神经元症状、颅神经麻痹、低血压、心动过缓以及呼吸和心脏骤停。复苏团队应做好处理这些并发症的准备。