Onguema Junior Rocyr Ibara, Raoul Florian, Marchais Aurelie, Amoura Alaa Al, Girodet Bertrand, Dacunka Mariane, Chapoutot Laurent, Maillier Bruno
Service de Cardiologie, Hôpital Simone Veil, Centre hospitalier de Troyes, France.
Service de Cardiologie, Hôpital Simone Veil, Centre hospitalier de Troyes, France.
Ann Cardiol Angeiol (Paris). 2023 Jun;72(3):101594. doi: 10.1016/j.ancard.2023.101594. Epub 2023 Apr 13.
Chronic Constrictive pericarditis (CCP) is a rare clinical entity that can pose diagnostic problems. Constrictive pericarditis is the final stage of a chronic inflammatory process characterized by fibrous thickening and calcification of the pericardium that impairs diastolic filling, reduces cardiac output, and ultimately leads to right heart failure and to atrial dilatation which can caused supravetricular arrythmia. Transthoracic echocardiography, computed tomography, and cardiac magnetic resonance imaging each can reveal severe diastolic dysfunction, increased pericardial thickness and calcifications. The gold standard for diagnosis is cardiac catheterization with analysis of intracavitary pressure curves, which are high and, in end diastole, equal in all chambers. CCP is the commun cause of recurrent heart failure. At present, idiopathic or viral pericarditis is the predominant cause of CP in the Western world, followed by postcardiotomy irritation and mediastinal irradiation. Tuberculosis is still a cause of pericarditis in developing countries and in immunosuppressed patient. We present a patient with symptomatic atrial fibrillation revealing chronic constrictive pericardis. He underwent to drug cardioversion before radical pericardiectomy and to date has made a good recovery without palpitations with a sinus rythm. The diagnosis of CP is often neglected by physicians, who usually attribute the symptoms to another disease process. This case show the difficulty in diagnosing, illustrated as well as the role of multimodality imaging and the excellent outcome of pericardiectomy for total recovery.
慢性缩窄性心包炎(CCP)是一种罕见的临床病症,可能会带来诊断难题。缩窄性心包炎是慢性炎症过程的终末期,其特征为心包纤维增厚和钙化,这会损害舒张期充盈,降低心输出量,并最终导致右心衰竭和心房扩张,进而引发室上性心律失常。经胸超声心动图、计算机断层扫描和心脏磁共振成像均可显示严重的舒张功能障碍、心包厚度增加和钙化。诊断的金标准是进行心导管检查并分析心腔内压力曲线,这些曲线在所有心腔中均较高且在舒张末期相等。CCP是复发性心力衰竭的常见原因。目前,特发性或病毒性心包炎是西方世界缩窄性心包炎的主要原因,其次是心脏手术后刺激和纵隔放疗。在发展中国家和免疫抑制患者中,结核病仍是心包炎的一个病因。我们报告一例有症状性心房颤动的患者,其被诊断为慢性缩窄性心包炎。在进行根治性心包切除术之前,他接受了药物复律,迄今为止恢复良好,没有心悸,心律为窦性。CP的诊断常常被医生忽视,他们通常将症状归因于另一种疾病过程。本病例展示了诊断的困难,以及多模态成像的作用和心包切除术实现完全康复的良好效果。