Juarez C P, Tso M O, van Heuven W A, Hayreh M S, Hayreh S S
Int Ophthalmol. 1986 May;9(2-3):89-101. doi: 10.1007/BF00159837.
The retinal changes following the simultaneous occlusion of retinal vessels were examined at an ultrastructural level. The preservation of endothelial cells that prevented leakage of blood and horseradish peroxidase tracer material in the surrounding retinal tissues explained the lack of hemorrhages observed clinically. Mueller cell death resulted in severe structural disorganization of the retina, with disruption of the inner limiting membrane, involvement of the outer retinal layers, and fleurette formation. The retinal macrocysts were derived from ischemic infarction.
在超微结构水平上检查了视网膜血管同时闭塞后的视网膜变化。内皮细胞的保存防止了血液和辣根过氧化物酶示踪物质在周围视网膜组织中的渗漏，这解释了临床上观察到的无出血现象。穆勒细胞死亡导致视网膜严重的结构紊乱，包括内界膜破坏、视网膜外层受累以及小花形成。视网膜大囊肿源自缺血性梗死。
