Uniwersytet Jagielloński, Wydział Zarządzania i Komunikacji Społecznej, Instytut Psychologii Stosowanej.
Uniwersytet Jagielloński, Wydział Polonistyki, Katedra Polonistycznej Edukacji Nauczycielskiej.
Psychiatr Pol. 2024 Aug 31;58(4):669-680. doi: 10.12740/PP/OnlineFirst/161676.
Aaron T. Beck's cognitive model of depression became the basis for the analysis of cognitive factors as significant correlates and predictors of depression. Psychological mechanisms of depression related to the depressive cognitive triad, the specificity of cognitive schemas and errors in cognitive processing determined the directions of further research on depression. The presented analysis of selected studies confirms and extends Beck's assumptions about the role of cognitive factors in the development and maintenance of depressive symptoms. The analysis of psychological, neuroimaging and biochemical studies provides a broader perspective of understanding both the etiology and mechanisms that sustain the symptoms of depression. The development of neuroimaging research has broadened the knowledge about the brain mechanisms of depression - the presented research combine the theoretical constructs of the cognitive concept of depression with the characteristics of the activity of brain structures and their functional connections. The following conclusions seem to be particularly important for clinical practice: 1) impairment of the cognitive inhibition function in depression reduces the control of negative automatic thoughts - strengthening working memory is important; 2) different brain mechanisms for processing rewards and punishments in people suffering from depression and in healthy people explain the formation of self-esteem - this knowledge can be an important element of psychoeducation in therapeutic work; 3) neuroimaging studies indicate neuronal correlates of cognitive distortions observed in depression - for further research on depression, it would be particularly important to monitor changes in brain activity in the course of its treatment.
阿伦·T·贝克(Aaron T. Beck)的抑郁症认知模型成为分析认知因素作为抑郁的重要相关因素和预测因素的基础。与抑郁性认知三联征、认知图式的特异性和认知加工错误相关的抑郁心理机制,决定了进一步研究抑郁的方向。对选定研究的分析证实并扩展了贝克关于认知因素在抑郁症状的发展和维持中的作用的假设。对心理、神经影像学和生物化学研究的分析提供了对抑郁的病因和维持症状的机制的更广泛的理解视角。神经影像学研究的发展拓宽了我们对抑郁的大脑机制的认识——目前的研究将抑郁的认知概念的理论结构与大脑结构的活动特征及其功能连接结合起来。以下结论似乎对临床实践特别重要:1)抑郁症患者认知抑制功能受损会降低对消极自动思维的控制——增强工作记忆很重要;2)抑郁症患者和健康人在处理奖励和惩罚方面的不同大脑机制解释了自尊的形成——这些知识可以成为治疗工作中心理教育的重要内容;3)神经影像学研究表明,抑郁中观察到的认知扭曲存在神经元相关性——对于进一步研究抑郁症,特别重要的是在治疗过程中监测大脑活动的变化。