Sun A Y, Boney F, Lee D Z
Neurochem Res. 1985 Feb;10(2):251-8. doi: 10.1007/BF00964571.
Electroacupuncture (EA) stimulation mediated the release of [3H]norepinephrine (NE) from synaptosomes prelabeled with [3H]NE. The pulse release of [3H]NE by EA stimulation was dependent on the presence of Ca2+. Treatment of rats with EA for 30 min at 4 Hz did not significantly alter the dopamine (DA) content in hypothalamus, cerebellum, pons, midbrain, and cerebral cortex regions, but the DA level was decreased by 20% in caudate nucleus. The NE level was found to increase by 43% in caudate nucleus and 38% in hypothalamus. The results indicate that only certain neuronal pathways are affected by the EA treatment, and that NE and DA may respond differently to such stimulation.
电针刺激介导了预先用[3H]去甲肾上腺素（NE）标记的突触体中[3H]NE的释放。电针刺激引起的[3H]NE脉冲式释放依赖于Ca2+的存在。以4Hz频率对大鼠进行30分钟的电针治疗，并未显著改变下丘脑、小脑、脑桥、中脑和大脑皮层区域的多巴胺（DA）含量，但尾状核中的DA水平降低了20%。发现尾状核中的NE水平增加了43%，下丘脑中增加了38%。结果表明，电针治疗仅影响某些神经通路，并且NE和DA对这种刺激的反应可能不同。
