病毒感染中的自然杀伤细胞：特刊社论
Natural Killer Cells in Viral Infection: Special Issue Editorial.
作者信息
Fang Min
机构信息
School of Life Sciences, Henan University, Kaifeng 475001, China.
出版信息
Viruses. 2025 Mar 10;17(3):391. doi: 10.3390/v17030391.
Abstract
NK cells are a critical cell population in innate immunity, playing an important role in antiviral immunity [...].
摘要
自然杀伤细胞是固有免疫中的关键细胞群体，在抗病毒免疫中发挥着重要作用[...]。
相似文献
1
Natural Killer Cells in Viral Infection: Special Issue Editorial.病毒感染中的自然杀伤细胞：特刊社论
Viruses. 2025 Mar 10;17(3):391. doi: 10.3390/v17030391.
2
Viral Evasion of Natural Killer Cell Activation.病毒对自然杀伤细胞激活的逃避
Viruses. 2016 Apr 12;8(4):95. doi: 10.3390/v8040095.
3
Roles of natural killer cells in antiviral immunity.自然杀伤细胞在抗病毒免疫中的作用。
Curr Opin Virol. 2016 Feb;16:15-23. doi: 10.1016/j.coviro.2015.10.008. Epub 2015 Nov 16.
4
Natural killer cell activation receptors in innate immunity to infection.天然免疫中感染的自然杀伤细胞激活受体
Microbes Infect. 2002 Dec;4(15):1513-21. doi: 10.1016/s1286-4579(02)00034-5.
5
Natural killer cell responses during viral infections: flexibility and conditioning of innate immunity by experience.自然杀伤细胞在病毒感染期间的反应：经验对先天免疫的灵活性和调节。
Curr Opin Virol. 2011 Dec;1(6):497-512. doi: 10.1016/j.coviro.2011.10.017.
6
Natural killer cell responses to viral infection.自然杀伤细胞对病毒感染的反应。
J Innate Immun. 2011;3(3):274-9. doi: 10.1159/000324176. Epub 2011 Mar 12.
7
Mutually assured destruction: the cold war between viruses and natural killer cells.相互确保毁灭：病毒与自然杀伤细胞之间的冷战。
Curr Opin Virol. 2019 Feb;34:130-139. doi: 10.1016/j.coviro.2019.02.005. Epub 2019 Mar 13.
8
Natural killer cells in antiviral immunity.自然杀伤细胞在抗病毒免疫中的作用。
Nat Rev Immunol. 2022 Feb;22(2):112-123. doi: 10.1038/s41577-021-00558-3. Epub 2021 Jun 11.
9
Natural killer cells: tolerance to self and innate immunity to viral infection and malignancy.自然杀伤细胞：对自身的耐受性和对病毒感染及恶性肿瘤的先天免疫。
Biol Blood Marrow Transplant. 2010 Jan;16(1 Suppl):S97-S105. doi: 10.1016/j.bbmt.2009.10.009. Epub 2009 Oct 14.
10
Control of human viral infections by natural killer cells.自然杀伤细胞对人类病毒感染的控制。
Annu Rev Immunol. 2013;31:163-94. doi: 10.1146/annurev-immunol-032712-100001. Epub 2013 Jan 3.
本文引用的文献
1
The combination of three CD4-induced antibodies targeting highly conserved Env regions with a small CD4-mimetic achieves potent ADCC activity.三种靶向高度保守 Env 区的 CD4 诱导型抗体与一个小的 CD4 模拟物的组合实现了有效的 ADCC 活性。
J Virol. 2024 Oct 22;98(10):e0101624. doi: 10.1128/jvi.01016-24. Epub 2024 Sep 9.
2
Metabolic requirements of CD160 expressing memory-like NK cells in Gram-negative bacterial infection.革兰氏阴性细菌感染中表达CD160的记忆样自然杀伤细胞的代谢需求。
Clin Transl Immunology. 2024 Jul 2;13(7):e1513. doi: 10.1002/cti2.1513. eCollection 2024.
3
Liver type 1 innate lymphoid cells develop locally via an interferon-γ-dependent loop.肝脏 1 型先天淋巴细胞通过依赖干扰素-γ的环在局部发育。
Science. 2021 Mar 26;371(6536). doi: 10.1126/science.aba4177.
4
Hepatitis C virus-induced natural killer cell proliferation involves monocyte-derived cells and the OX40/OX40L axis.丙型肝炎病毒诱导的自然杀伤细胞增殖涉及单核细胞衍生细胞和 OX40/OX40L 轴。
J Hepatol. 2018 Mar;68(3):421-430. doi: 10.1016/j.jhep.2017.10.021. Epub 2017 Nov 1.
5
IFN-γ production by human natural killer cells in response to HCV-infected hepatoma cells is dependent on accessory cells.人自然杀伤细胞针对 HCV 感染的肝癌细胞产生 IFN-γ 依赖于辅助细胞。
J Hepatol. 2013 Sep;59(3):442-9. doi: 10.1016/j.jhep.2013.04.022. Epub 2013 May 7.
6
Liver-resident NK cells confer adaptive immunity in skin-contact inflammation.肝脏驻留 NK 细胞在皮肤接触性炎症中赋予适应性免疫。
J Clin Invest. 2013 Apr;123(4):1444-56. doi: 10.1172/JCI66381. Epub 2013 Mar 25.
7
Expansion of a unique CD57⁺NKG2Chi natural killer cell subset during acute human cytomegalovirus infection.急性人巨细胞病毒感染期间独特的 CD57⁺NKG2Chi 自然杀伤细胞亚群的扩增。
Proc Natl Acad Sci U S A. 2011 Sep 6;108(36):14725-32. doi: 10.1073/pnas.1110900108. Epub 2011 Aug 8.
8
Adaptive immune features of natural killer cells.自然杀伤细胞的适应性免疫特征。
Nature. 2009 Jan 29;457(7229):557-61. doi: 10.1038/nature07665. Epub 2009 Jan 11.
9
T cell- and B cell-independent adaptive immunity mediated by natural killer cells.由自然杀伤细胞介导的不依赖T细胞和B细胞的适应性免疫。
Nat Immunol. 2006 May;7(5):507-16. doi: 10.1038/ni1332. Epub 2006 Apr 16.
10
Epistatic interaction between KIR3DS1 and HLA-B delays the progression to AIDS.KIR3DS1与HLA - B之间的上位性相互作用延缓了艾滋病的进展。
Nat Genet. 2002 Aug;31(4):429-34. doi: 10.1038/ng934. Epub 2002 Jul 22.
Zotero 插件