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细胞融合的生化研究。II. 脂质改变对融合反应的控制。

Biochemical studies on cell fusion. II. Control of fusion response by lipid alteration.

作者信息

Roos D S, Choppin P W

出版信息

J Cell Biol. 1985 Oct;101(4):1591-8. doi: 10.1083/jcb.101.4.1591.

Abstract

The preceding communication (Roos, D.S. and P.W. Choppin, 1985, J. Cell Biol. 101:1578-1590) described the lipid composition of a series of mouse fibroblast cell lines which vary in susceptibility to the fusogenic effects of polyethylene glycol (PEG). Two alterations in lipid content were found to be directly correlated with resistance to PEG-induced cell fusion: increases in fatty acyl chain saturation, and the elevation of neutral glycerides, including an unusual ether-linked compound. In this study, we have probed the association between lipid composition and cell fusion through the use of fatty acid supplements to the cellular growth medium, and show that the fusibility of cells can be controlled by altering their acyl chain composition. The parental Clone 1D cells contain moderately unsaturated fatty acids with a ratio of saturates to polyunsaturates (S/P) approximately 1 and fuse virtually to completion following a standard PEG treatment. By contrast, the lipids of a highly fusion-resistant mutant cell line, F40, are highly saturated (S/P approximately 4). When the S/P ratio of Clone 1D cells was increased to approximate that normally found in F40 cells by growth in the presence of high concentrations of saturated fatty acids, they became highly resistant to PEG. Reduction of the S/P ratio of F40 cells by growth in cis-polyunsaturated fatty acids rendered them susceptible to fusion. Cell lines F8, F16, etc., which are normally intermediate between Clone 1D and F40 in both lipid composition and fusion response, can be altered in either direction (towards either increased or decreased susceptibility to fusion) by the addition of appropriate fatty acids to the growth medium. Although trans-unsaturated fatty acids have phase-transition temperatures roughly similar to saturated compounds, and might therefore be expected to affect membrane fluidity in a similar manner, trans-unsaturated fatty acids exerted the same effect as cis-unsaturates on the control of PEG-induced cell fusion. This observation suggests that the control of cell fusion by alteration of fatty acid content is not due to changes in membrane fluidity, and thus that the fatty acids are involved in some other way in the modulation of cell fusion.

摘要

前文(Roos, D.S. 和 P.W. Choppin,1985 年,《细胞生物学杂志》101:1578 - 1590)描述了一系列对聚乙二醇(PEG)的融合效应敏感性不同的小鼠成纤维细胞系的脂质组成。发现脂质含量的两种变化与对 PEG 诱导的细胞融合的抗性直接相关:脂肪酰链饱和度增加,以及中性甘油酯升高,包括一种不寻常的醚连接化合物。在本研究中,我们通过向细胞生长培养基中添加脂肪酸来探究脂质组成与细胞融合之间的关联,并表明细胞的融合性可以通过改变其酰链组成来控制。亲本克隆 1D 细胞含有中度不饱和脂肪酸,饱和脂肪酸与多不饱和脂肪酸的比例(S/P)约为 1,在标准 PEG 处理后几乎完全融合。相比之下,高度抗融合的突变细胞系 F40 的脂质高度饱和(S/P 约为 4)。当克隆 1D 细胞在高浓度饱和脂肪酸存在下生长,使其 S/P 比值增加到与 F40 细胞中通常发现的比值相近时,它们对 PEG 变得高度抗性。通过在顺式多不饱和脂肪酸中生长来降低 F40 细胞的 S/P 比值,使其易于融合。细胞系 F8、F16 等,其脂质组成和融合反应通常介于克隆 1D 和 F40 之间,通过向生长培养基中添加适当的脂肪酸,可以在两个方向上改变(朝着对融合的敏感性增加或降低)。尽管反式不饱和脂肪酸的相变温度与饱和化合物大致相似,因此可能预期以类似方式影响膜流动性,但反式不饱和脂肪酸在控制 PEG 诱导的细胞融合方面与顺式不饱和脂肪酸具有相同的效果。这一观察结果表明,通过改变脂肪酸含量来控制细胞融合不是由于膜流动性的变化,因此脂肪酸以某种其他方式参与细胞融合的调节。

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