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Induction of a ouabain-sensitive ATPase defect by uremic plasma.

作者信息

Cole C H, Balfe J W, Welt L G

出版信息

Trans Assoc Am Physicians. 1968;81:213-20.

PMID:4238478
Abstract
摘要

相似文献

1
Induction of a ouabain-sensitive ATPase defect by uremic plasma.尿毒症血浆诱导哇巴因敏感的ATP酶缺陷
Trans Assoc Am Physicians. 1968;81:213-20.
2
Membrane transport defect: the sick cell.膜转运缺陷：病变细胞。
Trans Assoc Am Physicians. 1967;80:217-26.
3
Characteristics of active sodium and potassium transport in erythrocytes in children with different stages of symptomatic uremia.
Acta Paediatr Scand. 1981;70(3):361-8. doi: 10.1111/j.1651-2227.1981.tb16564.x.
4
[Effect of inorganic phosphates, urea and creatinine on ATPase activity in erythrocyte shadows obtained from healthy subjects and uremic patients].[无机磷酸盐、尿素和肌酐对健康受试者及尿毒症患者红细胞影中ATP酶活性的影响]
Pol Tyg Lek. 1975 Jul 28;30(30):1245-8.
5
[Sodium and potassium-dependent adenosine triphosphatase activity in the erythrocytes of some species of birds].[某些鸟类红细胞中钠钾依赖型三磷酸腺苷酶活性]
Boll Soc Ital Biol Sper. 1965 Sep 15;41(17):999-1002.
6
Functional and metabolic studies on red blood cell sodium transport in chronic uremia.慢性尿毒症患者红细胞钠转运的功能与代谢研究
Nephron. 1976;16(5):344-58. doi: 10.1159/000180621.
7
Red cell stromal adenosine triphosphatase (ATPase) of newborn infants.新生儿红细胞基质三磷酸腺苷酶（ATP酶）
Pediatr Res. 1969 Mar;3(2):105-12. doi: 10.1203/00006450-196903000-00001.
8
[Effect of dialysis on Na+/K+-ATP-ase activity in uremic children].[透析对尿毒症患儿钠钾-ATP酶活性的影响]
Orv Hetil. 1996 Oct 6;137(40):2193-6.
9
Sodium outflux and ATPase activity in human and rabbit erythrocytes.
J Appl Physiol. 1971 Jul;31(1):161-3. doi: 10.1152/jappl.1971.31.1.161.
10
Inhibition of brain sodium-potassium ATPase in uremic rats.尿毒症大鼠脑钠钾ATP酶的抑制作用。
J Lab Clin Med. 1972 Jul;80(1):71-8.

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1
Altered distribution of digoxin in renal failure--a cause of digoxin toxicity?肾衰竭时地高辛分布改变——地高辛中毒的一个原因？
Br J Clin Pharmacol. 1976 Dec;3(6):1045-51. doi: 10.1111/j.1365-2125.1976.tb00356.x.
2
Mechanism of alteration of sodium potassium pump of erythrocytes from patients with chronic renal failure.慢性肾功能衰竭患者红细胞钠钾泵改变的机制
J Clin Invest. 1984 Nov;74(5):1811-20. doi: 10.1172/JCI111600.
3
Erythrocyte Na,K pump in uremia. Acute correction of a transport defect by hemodialysis.尿毒症中的红细胞钠钾泵。通过血液透析对转运缺陷进行急性纠正。
J Clin Invest. 1984 Aug;74(2):581-8. doi: 10.1172/JCI111455.
4
Alterations in cellular membrane function during hemorrhagic shock in primates.灵长类动物失血性休克期间细胞膜功能的改变。
Ann Surg. 1972 Sep;176(3):288-95. doi: 10.1097/00000658-197209000-00004.
5
Abnormal sodium transport in synaptosomes from brain of uremic rats.尿毒症大鼠脑突触体中钠转运异常。
J Clin Invest. 1985 Jun;75(6):2014-23. doi: 10.1172/JCI111920.
6
Abnormal cation transport in uremia. Mechanisms in adipocytes and skeletal muscle from uremic rats.尿毒症中的异常阳离子转运。尿毒症大鼠脂肪细胞和骨骼肌中的机制。
J Clin Invest. 1988 Apr;81(4):1197-203. doi: 10.1172/JCI113435.
7
Erythrocyte sodium transport in dialyzed uremic patients.透析尿毒症患者的红细胞钠转运
Korean J Intern Med. 1989 Jan;4(1):9-17. doi: 10.3904/kjim.1989.4.1.9.
8
Monitoring digoxin therapy: II. Determinants of the apparent volume of distribution.地高辛治疗监测：II. 表观分布容积的决定因素。
Br J Clin Pharmacol. 1977 Apr;4(2):223-7. doi: 10.1111/j.1365-2125.1977.tb00698.x.