传染性无丙种球蛋白血症：无丙种球蛋白血症细胞移植导致免疫缺陷的传播。
Infectious agammaglobulinemia: transmission of immunodeficiency with grafts of agammaglobulinemic cells.
作者信息
Blaese R M, Weiden P L, Koski I, Dooley N
出版信息
J Exp Med. 1974 Oct 1;140(4):1097-1101. doi: 10.1084/jem.140.4.1097.
Abstract
Agammaglobulinemia induced in chickens by surgical bursectomy and irradiation at hatching is transmissible at 4 mo of age to sublethally irradiated normal chickens by bone marrow transplantation. The immunodeficiency which develops in the recipients persists for life and is characterized by inability to produce antibody and by progressive loss of detectable serum IgM and IgG.
摘要
通过在孵化时进行手术切除法氏囊和照射诱导鸡产生的无丙种球蛋白血症，在4月龄时通过骨髓移植可传播给经亚致死剂量照射的正常鸡。受体中出现的免疫缺陷会持续终生，其特征是无法产生抗体以及可检测到的血清IgM和IgG逐渐丧失。
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Infectious agammaglobulinemia: transmission of immunodeficiency with grafts of agammaglobulinemic cells.传染性无丙种球蛋白血症：无丙种球蛋白血症细胞移植导致免疫缺陷的传播。
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本文引用的文献
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IMMUNOLOGIC INCOMPETENCE OF IMMUNOLOGICALLY RUNTED ANIMALS.免疫矮小症动物的免疫功能不全。
J Exp Med. 1964 Feb 1;119(2):211-24. doi: 10.1084/jem.119.2.211.
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The thymus and the cellular basis of immunity.胸腺与免疫的细胞基础。
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Thymus and antigen-reactive cells.胸腺与抗原反应性细胞。
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Infectious immunological tolerance.感染性免疫耐受
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Migration of bone marrow cells to the bursa of Fabricius and the spleen in the chicken.骨髓细胞向鸡法氏囊和脾脏的迁移。
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In vitro studies of allotype suppression in mice.小鼠同种异型抑制的体外研究。
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Active suppression of immunoglobulin allotype synthesis. II. Transfer of suppressing factor with spleen cells.免疫球蛋白同种异型合成的主动抑制。II. 抑制因子与脾细胞的转移。
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Ontogeny of bursal function in chicken. I. Embryonic stem cell for humoral immunity.鸡法氏囊功能的个体发生。I. 体液免疫的胚胎干细胞。
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