酒精性心肌病。II. 乙醛对心脏微粒体蛋白质合成的抑制作用。 - Suppr

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超能文献

Alcoholic cardiomyopathy. II. The inhibition of cardiac microsomal protein synthesis by acetaldehyde.

作者信息

Schreiber S S, Oratz M, Rothschild M A, Reff F, Evans C

出版信息

J Mol Cell Cardiol. 1974 Apr;6(2):207-13. doi: 10.1016/0022-2828(74)90023-6.

DOI:10.1016/0022-2828(74)90023-6

PMID:4828692

Abstract

摘要

相似文献

Alcoholic cardiomyopathy. II. The inhibition of cardiac microsomal protein synthesis by acetaldehyde.酒精性心肌病。II. 乙醛对心脏微粒体蛋白质合成的抑制作用。

J Mol Cell Cardiol. 1974 Apr;6(2):207-13. doi: 10.1016/0022-2828(74)90023-6.

Ethanol, acetaldehyde, and myocardial protein synthesis.乙醇、乙醛与心肌蛋白合成

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Effect of acute overload on protein synthesis in cardiac muscle microsomes.

Am J Physiol. 1967 Dec;213(6):1552-5. doi: 10.1152/ajplegacy.1967.213.6.1552.

Alcoholic cardiomyopathy: the effect of ethanol and acetaldehyde on cardiac protein synthesis.酒精性心肌病：乙醇和乙醛对心脏蛋白质合成的影响。

Recent Adv Stud Cardiac Struct Metab. 1975;7:431-42.

Ethanol and cardiac protein synthesis.乙醇与心脏蛋白质合成

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The effect of ethanol and acetaldehyde on the metabolism and vascular resistance of the perfused heart.

Can J Biochem. 1971 Feb;49(2):227-33. doi: 10.1139/o71-033.

In vitro inhibition of protein synthesis in rat liver as a consequence of ethanol metabolism.乙醇代谢导致大鼠肝脏蛋白质合成的体外抑制。

Biochim Biophys Acta. 1974 Sep 27;366(1):101-8. doi: 10.1016/0005-2787(74)90322-0.

Inhibition of cardiac protein synthesis by prolonged ethanol administration.长期给予乙醇对心脏蛋白质合成的抑制作用。

Res Commun Chem Pathol Pharmacol. 1979 Jul;25(1):89-102.

Myosin, myoglobin, and collagen synthesis in acute cardiac overload.急性心脏负荷过重时的肌球蛋白、肌红蛋白和胶原蛋白合成

Am J Physiol. 1970 Aug;219(2):481-6. doi: 10.1152/ajplegacy.1970.219.2.481.

[Pathogenesis of alcoholic cardiomyopathy].

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引用本文的文献

The positive relationship between alcohol and heart disease in eastern Europe: potential physiological mechanisms.东欧地区酒精与心脏病之间的正向关系：潜在的生理机制

J R Soc Med. 1998 Aug;91(8):402-7. doi: 10.1177/014107689809100802.

High intracolonic acetaldehyde values produced by a bacteriocolonic pathway for ethanol oxidation in piglets.仔猪体内通过细菌结肠途径进行乙醇氧化产生的高结肠内乙醛值。

Gut. 1996 Jul;39(1):100-4. doi: 10.1136/gut.39.1.100.

Ultrastructural morphometric study on the rat heart after chronic ethanol feeding.慢性乙醇喂养后大鼠心脏的超微结构形态计量学研究。

Virchows Arch A Pathol Anat Histol. 1980;389(1):59-77. doi: 10.1007/BF00428668.

Effect of chronic ethanol treatment on peroxisomal acyl-CoA oxidase activity and lipid peroxidation in rat liver and heart.慢性乙醇处理对大鼠肝脏和心脏中过氧化物酶体酰基辅酶A氧化酶活性及脂质过氧化的影响。

Experientia. 1987 May 15;43(5):580-1. doi: 10.1007/BF02143593.

Role of macrophages in the pathogenesis of alcohol induced tissue damage.巨噬细胞在酒精诱导的组织损伤发病机制中的作用。

Br Med J (Clin Res Ed). 1987 May 2;294(6580):1137-9. doi: 10.1136/bmj.294.6580.1137.

Correlations between serum proteins modified by acetaldehyde and biochemical variables in heavy drinkers.乙醛修饰的血清蛋白与重度饮酒者生化指标之间的相关性。

J Clin Pathol. 1989 Mar;42(3):295-9. doi: 10.1136/jcp.42.3.295.

Protective action of ascorbic acid and sulfur compounds against acetaldehyde toxicity: implications in alcoholism and smoking.抗坏血酸和硫化合物对乙醛毒性的保护作用：对酗酒和吸烟的影响。

Agents Actions. 1975 May;5(2):164-73. doi: 10.1007/BF02027359.

Alcoholic liver injury.酒精性肝损伤

Proc R Soc Med. 1977 May;70(5):333-7. doi: 10.1177/003591577707000510.

Increased myocardial catalase in rats fed ethanol.喂食乙醇的大鼠心肌过氧化氢酶增加。

Am J Pathol. 1979 Aug;96(2):373-90.

[The regulation of protein synthesis in heart muscle. Biochemical data, stimulative and inhibitory factors and their clinical significance (author's transl)].[心肌中蛋白质合成的调节。生化数据、刺激和抑制因素及其临床意义（作者译）]

Klin Wochenschr. 1979 Jun 1;57(11):541-53. doi: 10.1007/BF01491133.

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