在一株缺乏切除修复功能的大肠杆菌中，产生嘧啶二聚体的突变在几个复制周期中持续存在。 - Suppr | 超能文献

The persistence through several replication cycles of mutation-producing pyrimidine dimers in a strain of Escherichia coli deficient in excision-repair.

作者信息

Bridges B A, Munson R J

出版信息

Biochem Biophys Res Commun. 1968 Mar 27;30(6):620-4. doi: 10.1016/0006-291x(68)90557-3.

DOI:10.1016/0006-291x(68)90557-3

Abstract

摘要

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1

The persistence through several replication cycles of mutation-producing pyrimidine dimers in a strain of Escherichia coli deficient in excision-repair.在一株缺乏切除修复功能的大肠杆菌中，产生嘧啶二聚体的突变在几个复制周期中持续存在。

Biochem Biophys Res Commun. 1968 Mar 27;30(6):620-4. doi: 10.1016/0006-291x(68)90557-3.

2

[The repair of radioinduced pyrimidine dimers in lambda bacteriophage DNA: participation of Escherichia coli uvr and rec genes and lambda red gene].[λ噬菌体DNA中辐射诱导嘧啶二聚体的修复：大肠杆菌uvr和rec基因以及λ red基因的参与]

Arch Int Physiol Biochim. 1969 May;77(2):397-9.

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Dark-recovery processes in Escherichia coli irradiated with ultraviolet light. 3. Effect of rec mutations on recovery of excision-deficient mutants of Escherichia coli K-12.紫外线照射大肠杆菌后的暗修复过程。3. rec突变对大肠杆菌K-12切除缺陷型突变体恢复的影响。

J Bacteriol. 1970 May;102(2):404-10. doi: 10.1128/jb.102.2.404-410.1970.

4

Distribution of pyrimidine dimers during postreplication repair in UV-irradiated excision-deficient cells of Escherichia coli K12.嘧啶二聚体在紫外线照射的大肠杆菌K12切除缺陷细胞复制后修复过程中的分布

Basic Life Sci. 1975;5A:317-20. doi: 10.1007/978-1-4684-2895-7_42.

5

Three loci in Escherichia coli K-12 that control the excision of pyrimidine dimers and certain other mutagen products from DNA.大肠杆菌K-12中控制从DNA切除嘧啶二聚体和某些其他诱变产物的三个基因座。

Genetics. 1966 Jun;53(6):1119-36. doi: 10.1093/genetics/53.6.1119.

6

New role for photoreversible pyrimidine dimers in induction of prototrophic mutations in excision-deficient Escherichia coli by UV light.光可逆嘧啶二聚体在紫外线诱导切除缺陷型大肠杆菌原养型突变中的新作用。

J Bacteriol. 1986 Jun;166(3):1141-3. doi: 10.1128/jb.166.3.1141-1143.1986.

7

Defective excision repair of pyrimidine dimers in the ultraviolet-sensitive Escherichia coli ras- mutant.紫外线敏感型大肠杆菌ras-突变体中嘧啶二聚体的切除修复缺陷。

J Bacteriol. 1970 Sep;103(3):552-9. doi: 10.1128/jb.103.3.552-559.1970.

8

Mechanism of repair of DNA in bacteriophage. II. Inability of ultraviolet-sensitive strains of bacteriophage in inducing an enzyme activity to excise pyrimidine dimers.噬菌体中DNA的修复机制。II. 紫外线敏感型噬菌体菌株诱导切除嘧啶二聚体的酶活性的无能。

J Mol Biol. 1970 Jan 28;47(2):243-55. doi: 10.1016/0022-2836(70)90343-8.

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Initial step of excision repair in Escherichia coli: replacement of defective function of uvr mutants by T4 endonuclease V.大肠杆菌切除修复的初始步骤：T4 核酸内切酶 V 替代 uvr 突变体的缺陷功能。

J Mol Biol. 1972 Sep 14;70(1):1-14. doi: 10.1016/0022-2836(72)90160-x.

10

Mechanism of repair of DNA in bacteriophage. I. Excision of pyrimidine dimers from ultraviolet-irradiated DNA by an extract of T4-infected cells.噬菌体中DNA的修复机制。I. 用T4感染细胞的提取物从紫外线照射的DNA中切除嘧啶二聚体。

J Mol Biol. 1970 Jan 28;47(2):231-42. doi: 10.1016/0022-2836(70)90342-6.

引用本文的文献

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Mutagenesis in Escherichia coli. II. Evidence for a common pathway for mutagenesis by ultraviolet light, ionizing radiation and thymine deprivation.大肠杆菌中的诱变作用。II. 紫外线、电离辐射和胸腺嘧啶缺失诱变的共同途径的证据。

Mol Gen Genet. 1968;103(3):266-73. doi: 10.1007/BF00273698.

2

Ultraviolet mutagenesis and its repair in an Escherichia coli strain containing a nonsense codon.含有无义密码子的大肠杆菌菌株中的紫外线诱变及其修复

Genetics. 1974 Dec;78(4):1035-49. doi: 10.1093/genetics/78.4.1035.

3

The influence of amino acid starvation on the UV reversibility of the strains of Escherichia coli B-r thy - trp - Hcr + and Escherichia coli B-r thy - trp - Hcr - .氨基酸饥饿对大肠杆菌B-r thy - trp - Hcr +菌株和大肠杆菌B-r thy - trp - Hcr -菌株紫外线可逆性的影响。

Folia Microbiol (Praha). 1972;17(4):274-80. doi: 10.1007/BF02880202.