Three loci in Escherichia coli K-12 that control the excision of pyrimidine dimers and certain other mutagen products from DNA.
作者信息
Howard-Flanders P, Boyce R P, Theriot L
出版信息
Genetics. 1966 Jun;53(6):1119-36. doi: 10.1093/genetics/53.6.1119.
Abstract
摘要
相似文献
1
Three loci in Escherichia coli K-12 that control the excision of pyrimidine dimers and certain other mutagen products from DNA.大肠杆菌K-12中控制从DNA切除嘧啶二聚体和某些其他诱变产物的三个基因座。
Genetics. 1966 Jun;53(6):1119-36. doi: 10.1093/genetics/53.6.1119.
2
Persistence of pyrimidine dimers during post-replication repair in ultraviolet light-irradiated Escherichia coli K12.紫外线照射的大肠杆菌K12在复制后修复过程中嘧啶二聚体的持续存在。
J Mol Biol. 1974 Jul 25;87(1):103-19. doi: 10.1016/0022-2836(74)90563-4.
3
The persistence through several replication cycles of mutation-producing pyrimidine dimers in a strain of Escherichia coli deficient in excision-repair.在一株缺乏切除修复功能的大肠杆菌中，产生嘧啶二聚体的突变在几个复制周期中持续存在。
Biochem Biophys Res Commun. 1968 Mar 27;30(6):620-4. doi: 10.1016/0006-291x(68)90557-3.
4
THE EXCISION OF THYMINE DIMERS FROM DNA, FILAMENT FORMATION AND SENSITIVITY TO ULTRAVIOLET LIGHT IN ESCHERICHIA COLI K-12.大肠杆菌K-12中DNA胸腺嘧啶二聚体的切除、丝状形成及对紫外线的敏感性
Mutat Res. 1964 Oct;106:219-26. doi: 10.1016/0027-5107(64)90002-8.
5
The excision of thymine dimer from the DNA of UV-irradiated E. coli 15 T-A-U during thymine deprivation.在胸腺嘧啶缺乏期间，从紫外线照射的大肠杆菌15 T-A-U的DNA中切除胸腺嘧啶二聚体。
Biochem Biophys Res Commun. 1964 Jul 27;16(5):489-96. doi: 10.1016/0006-291x(64)90381-x.
6
Genetic and physiological factors relating to excision-repair in Escherichia coli.与大肠杆菌切除修复相关的遗传和生理因素。
Johns Hopkins Med J. 1972;1:14-33.
7
Excision of pyrimidine dimers in toluene-treated Escherichia coli.甲苯处理的大肠杆菌中嘧啶二聚体的切除
J Bacteriol. 1976 Jan;125(1):220-4. doi: 10.1128/jb.125.1.220-224.1976.
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THE DISAPPEARANCE OF THYMINE DIMERS FROM DNA: AN ERROR-CORRECTING MECHANISM.胸腺嘧啶二聚体从DNA中的消失：一种纠错机制。
Proc Natl Acad Sci U S A. 1964 Feb;51(2):226-31. doi: 10.1073/pnas.51.2.226.
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Repair defect mutants of Proteus mirabilis. II. Excision of pyrimidine dimers from the DNA of ultraviolet-irradiated P. mirabilis wildtype and UV-sensitive mutants.奇异变形杆菌的修复缺陷突变体。II. 从紫外线照射的奇异变形杆菌野生型和紫外线敏感突变体的DNA中切除嘧啶二聚体。
Mol Gen Genet. 1973 Aug 17;124(3):259-68. doi: 10.1007/BF00293097.
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本文引用的文献
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Bacteriocidal action of mitomycin C.丝裂霉素C的杀菌作用。
Biochim Biophys Acta. 1961 Oct 14;53:132-49. doi: 10.1016/0006-3002(61)90800-9.
2
THE EXCISION OF THYMINE DIMERS FROM DNA, FILAMENT FORMATION AND SENSITIVITY TO ULTRAVIOLET LIGHT IN ESCHERICHIA COLI K-12.大肠杆菌K-12中DNA胸腺嘧啶二聚体的切除、丝状形成及对紫外线的敏感性
Mutat Res. 1964 Oct;106:219-26. doi: 10.1016/0027-5107(64)90002-8.
3
RELATIONSHIP BETWEEN ULTRAVIOLET SENSITIVITY AND ABILITY TO PROPAGATE ULTRAVIOLET-IRRADIATED BACTERIOPHAGE.紫外线敏感性与繁殖紫外线照射过的噬菌体的能力之间的关系。
J Bacteriol. 1964 Nov;88(5):1283-7. doi: 10.1128/jb.88.5.1283-1287.1964.
4
THE GENETIC MAP OF ESCHERICHIA COLI K-12.大肠杆菌K-12的遗传图谱
Genetics. 1964 Oct;50(4):659-77. doi: 10.1093/genetics/50.4.659.
5
PHYSICAL CHANGES AND MUTAGENESIS.物理变化与诱变作用
J Cell Comp Physiol. 1964 Oct;64:SUPPL 1:51-68.
6
GENE TRANSFER BY F' STRAINS OF ESCHERICHIA COLI K-12. I. DELAY IN INITIATION OF CHROMOSOME TRANSFER.大肠杆菌K-12的F'菌株介导的基因转移。I. 染色体转移起始的延迟
J Bacteriol. 1963 Jun;85(6):1394-401. doi: 10.1128/jb.85.6.1394-1401.1963.
7
Disappearance of thymine photodimer in ultraviolet irradiated DNA upon treatment with a photoreactivating enzyme from baker's yeast.用来自面包酵母的光复活酶处理后，紫外线照射的DNA中胸腺嘧啶光二聚体的消失
Biochem Biophys Res Commun. 1962 Apr 20;7:237-40. doi: 10.1016/0006-291x(62)90181-x.
8
The gene-controlled radiation sensitivity in Escherichia coli.大肠杆菌中基因控制的辐射敏感性。
Biochim Biophys Acta. 1963 Feb 26;68:263-70. doi: 10.1016/0006-3002(63)90141-0.
9
A method for selecting radiation-sensitive mutants of Escherichia coli.一种筛选大肠杆菌辐射敏感突变体的方法。
Genetics. 1962 Sep;47(9):1219-24. doi: 10.1093/genetics/47.9.1219.
10
A genetic locus in E. coli K12 that controls the reactivation of UV-photoproducts associated with thymine in DNA.大肠杆菌K12中一个控制与DNA中胸腺嘧啶相关的紫外线光产物再活化的基因座。
Proc Natl Acad Sci U S A. 1962 Dec 15;48(12):2109-15. doi: 10.1073/pnas.48.12.2109.
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