Sunshine M G, Kelly B
J Bacteriol. 1971 Nov;108(2):695-704. doi: 10.1128/jb.108.2.695-704.1971.
A series of independent Escherichia coli K eductants has been isolated and tested to determine the extent of their deletions. The deletions cover the P2 prophage in location H, the his operon, a suppressor of the recBC phenotype (sbcB), the gene for gluconate-6-phosphate dehydrogenase (gnd), a locus involved in cell wall synthesis (rfb), and in some cases all or part of genes involved in methylgalactoside uptake (mglP). One end of the deletion, the P2 prophage end, appears to be the same for all eductants. The other end, however, can be located before, within, and after the mglP locus.
已分离出一系列独立的大肠杆菌K诱变体，并对其进行测试以确定缺失的程度。这些缺失覆盖了位置H的P2原噬菌体、组氨酸操纵子、recBC表型的一个抑制子（sbcB）、6-磷酸葡萄糖酸脱氢酶基因（gnd）、一个参与细胞壁合成的位点（rfb），在某些情况下还覆盖了参与甲基半乳糖苷摄取的全部或部分基因（mglP）。所有诱变体的缺失一端，即P2原噬菌体末端，似乎是相同的。然而，另一端可以位于mglP位点之前、之中和之后。
