Immune lysis of normal human and paroxysmal nocturnal hemoglobinuria (PNH) red blood cells. II. The role of complement components in the increased sensitivity of PNH red cells to immune lysis.
作者信息
Rosse W F, Dacie J V
出版信息
J Clin Invest. 1966 May;45(5):749-57. doi: 10.1172/JCI105389.
Abstract
摘要
相似文献
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Immune lysis of normal human and paroxysmal nocturnal hemoglobinuria (PNH) red blood cells. II. The role of complement components in the increased sensitivity of PNH red cells to immune lysis.正常人及阵发性睡眠性血红蛋白尿（PNH）红细胞的免疫溶解。II. 补体成分在PNH红细胞对免疫溶解敏感性增加中的作用。
J Clin Invest. 1966 May;45(5):749-57. doi: 10.1172/JCI105389.
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Complement lysis of human erythrocytes. Differeing susceptibility of two types of paroxysmal nocturnal hemoglobinuria cells to C5b-9.人红细胞的补体溶解。两种阵发性夜间血红蛋白尿细胞对C5b-9的不同敏感性。
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Immune lysis of normal human and paroxysmal nocturnal hemoglobinuria (PNH) red blood cells. I. The sensitivity of PNH red cells to lysis by complement and specific antibody.正常人及阵发性睡眠性血红蛋白尿症（PNH）红细胞的免疫溶解作用。I. PNH红细胞对补体和特异性抗体介导的溶解作用的敏感性。
J Clin Invest. 1966 May;45(5):736-48. doi: 10.1172/JCI105388.
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Paroxysmal nocturnal hemoglobinuria type III. Lack of an erythrocyte membrane protein restricting the lysis by C5b-9.III型阵发性夜间血红蛋白尿。缺乏限制C5b-9介导红细胞溶解的红细胞膜蛋白。
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[Evaluation and quantification of complement-sensitive red cells in PNH and aplastic anemia-PNH syndrome].[阵发性睡眠性血红蛋白尿症及再生障碍性贫血-阵发性睡眠性血红蛋白尿症综合征中补体敏感红细胞的评估与定量分析]
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Serum-red cell interactions at low ionic strength: erythrocyte complement coating and hemolysis of paroxysmal nocturnal hemoglobinuria cells.低离子强度下的血清-红细胞相互作用：阵发性夜间血红蛋白尿症细胞的红细胞补体包被与溶血
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Mechanism of complement-mediated activation of human blood platelets in vitro: comparison of normal and paroxysmal nocturnal hemoglobinuria platelets.补体介导的人血小板体外激活机制：正常血小板与阵发性夜间血红蛋白尿血小板的比较
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Affected erythrocytes of patients with paroxysmal nocturnal hemoglobinuria are deficient in the complement regulatory protein, decay accelerating factor.阵发性夜间血红蛋白尿患者的受累红细胞缺乏补体调节蛋白衰变加速因子。
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Studies of paroxysmal nocturnal hemoglobinuria erythrocytes: increased lysis and lipid peroxide formation by hydrogen peroxide.阵发性夜间血红蛋白尿红细胞的研究：过氧化氢导致细胞溶解增加及脂质过氧化物生成
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Detection of complement components on unlysed erythrocytes from acid hemolysis and thrombin test reactions in paroxysmal nocturnal hemoglobinuria.阵发性夜间血红蛋白尿中酸溶血和凝血酶试验反应未溶血红细胞上补体成分的检测
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Immune lysis of normal human and paroxysmal nocturnal hemoglobinuria (PNH) red blood cells. I. The sensitivity of PNH red cells to lysis by complement and specific antibody.正常人及阵发性睡眠性血红蛋白尿症（PNH）红细胞的免疫溶解作用。I. PNH红细胞对补体和特异性抗体介导的溶解作用的敏感性。
J Clin Invest. 1966 May;45(5):736-48. doi: 10.1172/JCI105388.
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Paroxysmal nocturnal haemoglobinuria and leukaemia.阵发性夜间血红蛋白尿症与白血病
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Mechanisms of immune lysis of the red cells in hereditary erythroblastic multinuclearity with a positive acidified serum test and paroxysmal nocturnal hemoglobinuria.伴有阳性酸化血清试验的遗传性成红细胞多核症及阵发性夜间血红蛋白尿中红细胞免疫溶解的机制
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THE HEMOLYSIS OF RED CELLS FROM PATIENTS WITH PAROXYSMAL NOCTURNAL HEMOGLOBINURIA BY PARTIALLY PURIFIED SUB-COMPONENTS OF THE THIRD COMPLEMENT COMPONENT.阵发性夜间血红蛋白尿症患者红细胞被补体第三成分部分纯化亚组分的溶血作用
J Clin Invest. 1965 Sep;44(9):1534-46. doi: 10.1172/JCI105260.
2
EXPERIMENTAL PRODUCTION OF COLD AGGLUTININS IN RABBITS.家兔体内冷凝集素的实验性产生
Blood. 1965 Sep;26:323-40.
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ACTION OF THE FIRST COMPONENT OF COMPLEMENT. ACTIVITATION OF C'1 TO C'1A IN THE HEMOLYTIC SYSTEM.补体第一成分的作用。溶血系统中C'1激活为C'1a 。
J Immunol. 1964 Jan;92:108-17.
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EFFECTS OF LOW IONIC STRENGTH ON IMMUNE HEMOLYSIS.低离子强度对免疫溶血的影响
J Immunol. 1963 Dec;91:826-32.
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Comparison of the sensitivity to agglutination and haemolysis by a high-titre cold antibody of the erythrocytes of normal subjects and of patients with a variety of blood diseases including paroxysmal nocturnal haemoglobinuria.正常受试者以及包括阵发性夜间血红蛋白尿在内的多种血液病患者的红细胞对高效价冷凝集素的凝集和溶血敏感性比较。
Br J Haematol. 1961 Jan;7:64-72. doi: 10.1111/j.1365-2141.1961.tb00320.x.
9
Immune lysis of normal human and paroxysmal nocturnal hemoglobinuria (PNH) red blood cells. I. The sensitivity of PNH red cells to lysis by complement and specific antibody.正常人及阵发性睡眠性血红蛋白尿症（PNH）红细胞的免疫溶解作用。I. PNH红细胞对补体和特异性抗体介导的溶解作用的敏感性。
J Clin Invest. 1966 May;45(5):736-48. doi: 10.1172/JCI105388.
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