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A possible protective role for sulphydryl compounds in acute alcoholic liver injury.

作者信息

Macdonald C M, Dow J, Moore M R

出版信息

Biochem Pharmacol. 1977 Aug 15;26(16):1529-31. doi: 10.1016/0006-2952(77)90428-2.

DOI:10.1016/0006-2952(77)90428-2
PMID:561607
Abstract
摘要

相似文献

1
A possible protective role for sulphydryl compounds in acute alcoholic liver injury.
Biochem Pharmacol. 1977 Aug 15;26(16):1529-31. doi: 10.1016/0006-2952(77)90428-2.
2
On the mechanism of the glutathione depletion induced in the liver by acute ethanol ingestion.关于急性摄入乙醇引起肝脏中谷胱甘肽耗竭的机制。
Subst Alcohol Actions Misuse. 1981;2(3):153-60.
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[Effect of acute alcoholic and acetaldehyde poisoning on lipid metabolism in the rat liver].[急性酒精和乙醛中毒对大鼠肝脏脂质代谢的影响]
Ukr Biokhim Zh (1999). 1999 Jan-Feb;71(1):95-7.
4
Effects of acute ethanol intake on liver morphology and metabolism.急性乙醇摄入对肝脏形态和代谢的影响。
Acta Pharmacol Toxicol (Copenh). 1977;41 Suppl 2:273-81.
5
The use of sulphydryl containing drugs and chelating agents to reduce renal irradiation during 203Hg chlormerodrin scintigraphy.在203汞氯汞丙脲闪烁扫描术中使用含巯基药物和螯合剂以减少肾脏辐射。
J Nucl Biol Med. 1968 Jul-Sep;12(3):121-7.
6
[Metabolism of liver proteins in ethanol poisoning].[乙醇中毒时肝脏蛋白质的代谢]
Postepy Hig Med Dosw. 1992;46(1):117-30.
7
Free radicals and not acetaldehyde influence the circulating levels of glutathione after acute or chronic alcohol abuse: in vivo and in vitro studies.自由基而非乙醛影响急性或慢性酒精滥用后谷胱甘肽的循环水平：体内和体外研究。
Ital J Gastroenterol Hepatol. 1997 Apr;29(2):168-73.
8
Inhibition of the actinomycin-induced nucleolar change by thioacetamide in mouse hepatic parenchymal cell.硫代乙酰胺对放线菌素诱导的小鼠肝实质细胞核仁变化的抑制作用。
Lab Invest. 1967 Dec;17(6):767-71.
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[Ethanol-induced changes in protein synthesis of mitochondria and microsomes of rat brain and liver].[乙醇诱导大鼠脑和肝脏线粒体及微粒体蛋白质合成的变化]
Boll Soc Ital Biol Sper. 1973 Sep 15;49(17):977-82.
10
Stronger Neo-Minophagen C, a glycyrrhizin-containing preparation, protects liver against carbon tetrachloride-induced oxidative stress in transgenic mice expressing the hepatitis C virus polyprotein.强力新美能C，一种含甘草酸的制剂，可保护表达丙型肝炎病毒多聚蛋白的转基因小鼠肝脏免受四氯化碳诱导的氧化应激。
Liver Int. 2007 Aug;27(6):845-53. doi: 10.1111/j.1478-3231.2007.01492.x.

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An opium alkaloid-papaverine ameliorates ethanol-induced hepatotoxicity: Diminution of oxidative stress.一种鸦片生物碱——罂粟碱可改善乙醇诱导的肝毒性：减轻氧化应激。
Indian J Clin Biochem. 2000 Jul;15(2):155-60. doi: 10.1007/BF02883745.
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Glutathione depletion and recovery after acute ethanol administration in the aging mouse.衰老小鼠急性乙醇给药后谷胱甘肽的消耗与恢复
Biochem Pharmacol. 2007 May 15;73(10):1613-21. doi: 10.1016/j.bcp.2007.01.033. Epub 2007 Jan 30.
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Effect of age on alteration of glutathione metabolism following chronic cigarette smoke inhalation in mice.年龄对小鼠长期吸入香烟烟雾后谷胱甘肽代谢改变的影响。
Lung. 1996;174(2):119-26. doi: 10.1007/BF00177705.
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Effect of ethanol on CHCl3 metabolism in hepatic microsomes from Osborne-Mendel rats.乙醇对奥斯本-孟德尔大鼠肝微粒体中三氯甲烷代谢的影响。
Environ Health Perspect. 1994 Nov;102 Suppl 9(Suppl 9):25-30. doi: 10.1289/ehp.94102s925.
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Effect of ethanol on glutathione concentration in isolated hepatocytes.乙醇对分离的肝细胞中谷胱甘肽浓度的影响。
Biochem J. 1980 May 15;188(2):549-52. doi: 10.1042/bj1880549.
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Increased loss and decreased synthesis of hepatic glutathione after acute ethanol administration. Turnover studies.急性乙醇给药后肝脏谷胱甘肽损失增加及合成减少。周转研究。
Biochem J. 1985 Feb 1;225(3):565-72. doi: 10.1042/bj2250565.
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Synergism of organic zinc salts and sulfhydryl compounds (thiols) in the protection of mice against acute ethanol toxicity, and protective effects of various metal salts.
Agents Actions. 1987 Jun;21(1-2):217-22. doi: 10.1007/BF01974945.