患有X连锁B淋巴细胞缺陷的小鼠中缺乏自身抗原特异性脾抑制细胞。
Lack of autoantigen-specific splenic suppressor cells in mice with an X-linked B-lymphocyte defect.
作者信息
Cooke A, Hutchings P, Marshall-Clarke S
出版信息
Immunology. 1980 Dec;41(4):815-8.
Abstract
Male and female progeny of a cross between CBA/N female and BALB/c male mice were tested for their ability to generate autoantigen-specific suppressor cells as a result of stimulation with crossreacting rat RBC. Male mice, hemizygous for the X-linked defect, were unable to generate these antigen-specific suppressor cells, whereas their female littermates behaved like the normal (CBA/Ca x BALB/c)F1 mice.
摘要
对CBA/N雌性小鼠和BALB/c雄性小鼠杂交产生的雄性和雌性后代进行测试，以检测它们在用交叉反应大鼠红细胞刺激后产生自身抗原特异性抑制细胞的能力。携带X连锁缺陷的半合子雄性小鼠无法产生这些抗原特异性抑制细胞，而它们的雌性同窝小鼠表现得与正常的（CBA/Ca×BALB/c）F1小鼠一样。
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Immunology. 1980 Jul;40(3):335-42.
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Immunology. 1968 Apr;14(4):599-600.
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Induction of red cell autoantibodies in normal mice.正常小鼠中红细胞自身抗体的诱导
Nat New Biol. 1973 Jun 13;243(128):213-4. doi: 10.1038/newbio243213a0.
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Autoimmune haemolytic anaemia induced in mice immunized with rat erythrocytes.用大鼠红细胞免疫小鼠诱导产生的自身免疫性溶血性贫血。
Clin Exp Immunol. 1974 Jun;17(2):319-27.
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The relationship between antigenic structure and the requirement for thymus-derived cells in the immune response.免疫反应中抗原结构与胸腺依赖性细胞需求之间的关系。
J Exp Med. 1971 Jul 1;134(1):103-19. doi: 10.1084/jem.134.1.103.
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X-linked B-lymphocyte immune defect in CBA/N mice. II. Studies of the mechanisms underlying the immune defect.CBA/N小鼠的X连锁B淋巴细胞免疫缺陷。II.免疫缺陷潜在机制的研究。
J Exp Med. 1975 Sep 1;142(3):637-50. doi: 10.1084/jem.142.3.637.
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