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Potentially lethal and DNA radiation damage: similarities in inhibition of repair by medium containing D2O and by hypertonic buffer.

作者信息

Ben-Hur E, Utsumi H, Elkind M M

出版信息

Radiat Res. 1980 Oct;84(1):25-34.

PMID:6256804
Abstract
摘要

相似文献

1
Potentially lethal and DNA radiation damage: similarities in inhibition of repair by medium containing D2O and by hypertonic buffer.
Radiat Res. 1980 Oct;84(1):25-34.
2
"Single-hit" potentially lethal damage: evidence of its repair in mammalian cells.“单次打击”潜在致死性损伤：其在哺乳动物细胞中修复的证据
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3
Deuterium oxide enhancement of Chinese hamster cell response to gamma radiation.
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4
Ionizing radiation induces two forms of interphase chromosome breaks in Chinese hamster ovary cells that rejoin with different kinetics and show different sensitivity to treatment in hypertonic medium or beta-araA.电离辐射在中国仓鼠卵巢细胞中诱导出两种间期染色体断裂形式，它们以不同的动力学重新连接，并且在高渗培养基或β-阿拉伯糖腺苷处理下表现出不同的敏感性。
Radiat Res. 1993 Nov;136(2):262-70.
5
Caffeine and D2O medium interact in affecting the expression of radiation-induced potentially lethal damage.咖啡因和重水介质在影响辐射诱导的潜在致死性损伤的表达方面相互作用。
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6
Cultured cells from a severe combined immunodeficient mouse have a slower than normal rate of repair of potentially lethal damage sensitive to hypertonic treatment.
Radiat Res. 1995 May;142(2):176-80.
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Alteration in thermal sensitivity of chinese hamster cells by D2O treatment.重水（D₂O）处理对中国仓鼠细胞热敏感性的影响
Radiat Res. 1982 Jun;90(3):644-8.
8
Tests of the double-strand break, lethal-potentially lethal and repair-misrepair models for mammalian cell survival using data for survival as a function of delayed-plating interval for log-phase Chinese hamster V79 cells.利用对数生长期中国仓鼠V79细胞存活数据作为延迟接种间隔的函数，对哺乳动物细胞存活的双链断裂、潜在致死-致死和修复-错配修复模型进行测试。
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Fixation of potentially lethal radiation damage in Chinese hamster cells by anisotonic solutions, polyamines, and DMSO.
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10
Hypertonic treatment does not affect the radiation yield of interphase chromosome breaks in DNA double-strand break repair-deficient xrs-5 cells.高渗处理不影响DNA双链断裂修复缺陷型xrs-5细胞中期染色体断裂的辐射产额。
Radiat Res. 1993 Aug;135(2):171-7.

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Recovery after exposure to near-ultraviolet light of cells containing 5-bromodeoxyuridine.暴露于含有5-溴脱氧尿苷的细胞的近紫外光后的恢复情况。
Biophys J. 1981 Jun;34(3):367-74. doi: 10.1016/S0006-3495(81)84855-2.
2
Inhibitors of poly (ADP-ribose) synthesis enhance radiation response by differentially affecting repair of potentially lethal versus sublethal damage.聚（ADP - 核糖）合成抑制剂通过差异性地影响潜在致死性损伤与亚致死性损伤的修复来增强辐射反应。
Br J Cancer Suppl. 1984;6:39-42.
3
Relationship between DNA repair and cell recovery: importance of competing biochemical and metabolic processes.
Radiat Environ Biophys. 1985;24(4):271-80. doi: 10.1007/BF01210934.
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A cell survival model with saturable repair after irradiation.一种照射后具有饱和修复的细胞存活模型。
Radiat Environ Biophys. 1987;26(1):63-79. doi: 10.1007/BF01211365.
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Are single or multiple mechanisms involved in radiation-induced mammalian cell killing?辐射诱导哺乳动物细胞死亡涉及单一机制还是多种机制？
Br J Cancer Suppl. 1987 Jun;8:24-31.
6
In vitro holding and PLD repair. I. On the contribution of mitotic non-quiescence in plateau-phase Chinese hamster V79 cells.体外培养与多聚磷脂酰肌醇降解修复。I. 关于中国仓鼠V79细胞平台期有丝分裂非静止的作用。
Radiat Environ Biophys. 1989;28(1):27-38. doi: 10.1007/BF01209720.