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Herpes simplex virus thymidine kinase expression in trigeminal ganglion infection: correlation of enzyme activity with ganglion virus titer and evidence of in vivo complementation.

作者信息

Tenser R B, Ressel S, Dunstan M E

出版信息

Virology. 1981 Jul 15;112(1):328-41. doi: 10.1016/0042-6822(81)90638-3.

DOI:10.1016/0042-6822(81)90638-3
PMID:6264689
Abstract
摘要

相似文献

1
Herpes simplex virus thymidine kinase expression in trigeminal ganglion infection: correlation of enzyme activity with ganglion virus titer and evidence of in vivo complementation.单纯疱疹病毒胸苷激酶在三叉神经节感染中的表达：酶活性与神经节病毒滴度的相关性及体内互补的证据
Virology. 1981 Jul 15;112(1):328-41. doi: 10.1016/0042-6822(81)90638-3.
2
Herpes simplex virus thymidine kinase expression in infection of the trigeminal ganglion.单纯疱疹病毒胸苷激酶在三叉神经节感染中的表达
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Trigeminal ganglion infection by thymidine kinase-negative mutants of herpes simplex virus.单纯疱疹病毒胸苷激酶阴性突变体引起的三叉神经节感染
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Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate.胸苷激酶阴性单纯疱疹病毒突变体可在小鼠三叉神经节中建立潜伏感染，但不会再激活。
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A low thymidine kinase-producing mutant of herpes simplex virus type 1 causes latent trigeminal ganglia infections in mice.单纯疱疹病毒1型的一种低胸苷激酶产生突变体在小鼠中引起潜伏性三叉神经节感染。
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Detection of latent thymidine kinase-deficient herpes simplex virus in trigeminal ganglia of mice using the polymerase chain reaction.利用聚合酶链反应检测小鼠三叉神经节中潜伏的胸苷激酶缺陷型单纯疱疹病毒。
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Failure of thymidine kinase-negative herpes simplex virus to reactivate from latency following efficient establishment.胸苷激酶阴性单纯疱疹病毒在有效建立潜伏感染后无法从潜伏状态重新激活。
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引用本文的文献

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J Virol. 2015 Aug;89(16):8383-91. doi: 10.1128/JVI.00468-15.
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Phosphorylation of a herpes simplex virus 1 dUTPase by a viral protein kinase, Us3, dictates viral pathogenicity in the central nervous system but not at the periphery.单纯疱疹病毒1型dUTP酶被病毒蛋白激酶Us3磷酸化，决定了病毒在中枢神经系统而非外周的致病性。
J Virol. 2014 Mar;88(5):2775-85. doi: 10.1128/JVI.03300-13. Epub 2013 Dec 18.
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Diverse herpes simplex virus type 1 thymidine kinase mutants in individual human neurons and Ganglia.
个体人类神经元和神经节中多样的单纯疱疹病毒1型胸苷激酶突变体。
J Virol. 2007 Jul;81(13):6817-26. doi: 10.1128/JVI.00166-07. Epub 2007 Apr 25.
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The African swine fever virus thymidine kinase gene is required for efficient replication in swine macrophages and for virulence in swine.非洲猪瘟病毒胸苷激酶基因是在猪巨噬细胞中高效复制及对猪具有毒力所必需的。
J Virol. 1998 Dec;72(12):10310-5. doi: 10.1128/JVI.72.12.10310-10315.1998.
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J Virol. 1993 Nov;67(11):6903-8. doi: 10.1128/JVI.67.11.6903-6908.1993.
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Molecular analysis of a neurovirulent herpes simplex virus type 2 strain with reduced thymidine kinase activity.一株胸苷激酶活性降低的神经毒性2型单纯疱疹病毒的分子分析
Arch Virol. 1993;131(1-2):61-73. doi: 10.1007/BF01379080.
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The pathogenicity of a US3 protein kinase-deficient mutant of herpes simplex virus type 2 in mice.2型单纯疱疹病毒US3蛋白激酶缺陷型突变体在小鼠中的致病性。
Arch Virol. 1993;133(3-4):259-73. doi: 10.1007/BF01313767.
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Thymidine plaque autoradiography of thymidine kinase-positive and thymidine kinase-negative herpesviruses.胸苷激酶阳性和胸苷激酶阴性疱疹病毒的胸苷斑块放射自显影术。
J Clin Microbiol. 1983 Jan;17(1):122-7. doi: 10.1128/jcm.17.1.122-127.1983.
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Cooperative effects between two acyclovir resistance loci in herpes simplex virus.单纯疱疹病毒中两个阿昔洛韦抗性位点之间的协同效应。
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Neurovirulence of herpes simplex virus type 1 depends on age in mice and thymidine kinase expression.1型单纯疱疹病毒的神经毒性取决于小鼠的年龄和胸苷激酶的表达。
Arch Virol. 1983;78(3-4):303-8. doi: 10.1007/BF01311326.