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At least two regions of the viral genome determine the oncogenic potential of avian leukosis viruses.
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Cancer induction by insertional mutagenesis: the role of viral genes in avian leukosis virus induced cancers.
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Region coding for subgroup specificity of envelope of avian retroviruses does not determine lymphomagenicity.
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Long terminal repeat (LTR) sequences, env, and a region near the 5' LTR influence the pathogenic potential of recombinants between Rous-associated virus types 0 and 1.
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Pathogenicity of avian leukosis viruses.
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5' avian leukosis virus sequences and osteopetrotic potential.
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Genetics of leukemogenesis by avian leukosis viruses.
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Avian leukosis virus-induced tumors have common proviral integration sites and synthesize discrete new RNAs: oncogenesis by promoter insertion.
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Risk of Mortality From Esophageal Cancer Among US Poultry Workers, 1950-2019.
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A recombinant avian leukosis virus subgroup j for directly monitoring viral infection and the selection of neutralizing antibodies.
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Detection and molecular characterization of J subgroup avian leukosis virus in wild ducks in China.
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A 205-nucleotide deletion in the 3' untranslated region of avian leukosis virus subgroup J, currently emergent in China, contributes to its pathogenicity.
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Molecular epidemiology of avian leukosis virus subgroup J in layer flocks in China.
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VBP and RelA regulate avian leukosis virus long terminal repeat-enhanced transcription in B cells.
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In vitro CD4+ lymphocyte transformation and infection in a rabbit model with a molecular clone of human T-cell lymphotrophic virus type 1.
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Unique long terminal repeat U3 sequences distinguish exogenous jaagsiekte sheep retroviruses associated with ovine pulmonary carcinoma from endogenous loci in the sheep genome.
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Multiple viral determinants contribute to pathogenicity of the acutely lethal simian immunodeficiency virus SIVsmmPBj variant.
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Comparison of 5' and 3' long terminal repeat promoter function in human immunodeficiency virus.
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