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Human parvovirus aplasia: case due to cross infection in a ward.人类细小病毒所致全血细胞减少症:病房内交叉感染病例
Br Med J (Clin Res Ed). 1984 Mar 3;288(6418):681. doi: 10.1136/bmj.288.6418.681.
2
Human parvovirus infection and aplastic crisis in hereditary spherocytosis.遗传性球形红细胞增多症中的人细小病毒感染与再生障碍危象
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[Aplastic crises in hereditary spherocytosis].[遗传性球形红细胞增多症中的再生障碍危象]
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[Aplastic crisis due to human parvovirus B19 infection in hereditary spherocytosis].[遗传性球形红细胞增多症中因人类细小病毒B19感染所致的再生障碍危象]
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Parvovirus infection in a family associated with aplastic crisis in an affected sibling pair with hereditary spherocytosis.一个家庭中的细小病毒感染与一对患遗传性球形红细胞增多症的患病同胞的再生障碍危象相关。
J Clin Pathol. 1984 Oct;37(10):1144-6. doi: 10.1136/jcp.37.10.1144.
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Six cases of hereditary spherocytosis revealed by human parvovirus infection.6例由人细小病毒感染引发的遗传性球形红细胞增多症
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Aplastic crisis associated with parvovirus B19 in an adult with hereditary spherocytosis.一名患有遗传性球形红细胞增多症的成年人中与细小病毒B19相关的再生障碍危象。
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[Case of hereditary spherocytic anemia with an aplastic attack due to parvovirus infection].[因细小病毒感染引发再生障碍危象的遗传性球形红细胞增多症病例]
Nihon Naika Gakkai Zasshi. 1994 Jul 10;83(7):1197-8.
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[A case of hereditary spherocytosis complicated with aplastic crisis induced by parvovirus infection].[一例遗传性球形红细胞增多症合并细小病毒感染所致再生障碍危象]
Nihon Naika Gakkai Zasshi. 1988 Apr;77(4):553-7. doi: 10.2169/naika.77.553.

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1
Parvovirus B19 infection in human pregnancy.人妊娠期间的细小病毒 B19 感染。
BJOG. 2011 Jan;118(2):175-86. doi: 10.1111/j.1471-0528.2010.02749.x. Epub 2010 Oct 13.
2
Human parvovirus B19.人细小病毒B19
Clin Microbiol Rev. 2002 Jul;15(3):485-505. doi: 10.1128/CMR.15.3.485-505.2002.
3
Parvovirus B19 infection.细小病毒B19感染
Eur J Clin Microbiol Infect Dis. 1996 Jan;15(1):10-29. doi: 10.1007/BF01586181.
4
Parvovirus infection causing red cell aplasia and leukopenia in rheumatoid arthritis.细小病毒感染在类风湿关节炎中导致红细胞再生障碍和白细胞减少。
Clin Rheumatol. 1994 Mar;13(1):129-31. doi: 10.1007/BF02229883.
5
Fifth disease: report of an outbreak.传染性红斑:一次疫情报告。
J R Coll Gen Pract. 1984 Oct;34(267):573-4.
6
Parvovirus infection in a family associated with aplastic crisis in an affected sibling pair with hereditary spherocytosis.一个家庭中的细小病毒感染与一对患遗传性球形红细胞增多症的患病同胞的再生障碍危象相关。
J Clin Pathol. 1984 Oct;37(10):1144-6. doi: 10.1136/jcp.37.10.1144.
7
An outbreak of erythema infectiosum associated with human parvovirus infection.一起与人类细小病毒感染相关的传染性红斑暴发。
J Hyg (Lond). 1984 Aug;93(1):85-93. doi: 10.1017/s0022172400060964.
8
Hereditary spherocytosis in sisters unmasked by parvovirus infection.细小病毒感染引发姐妹二人的遗传性球形红细胞增多症
Postgrad Med J. 1987 Jan;63(735):49-50. doi: 10.1136/pgmj.63.735.49.
9
B19 parvovirus infection causing aplastic crisis in 3 out of 5 family members with hereditary spherocytosis.B19细小病毒感染在5名遗传性球形红细胞增多症家庭成员中的3人身上引发再生障碍性危象。
Ir J Med Sci. 1990 Jun;159(6):182-4. doi: 10.1007/BF02937239.
10
Erythema infectiosum and pregnancy-related complications.传染性红斑与妊娠相关并发症。
CMAJ. 1990 Nov 1;143(9):849-58.

本文引用的文献

1
Human serum "parvovirus": a specific cause of aplastic crisis in children with hereditary spherocytosis.人血清“细小病毒”:遗传性球形红细胞增多症患儿再生障碍危象的特定病因。
J Pediatr. 1983 May;102(5):720-2. doi: 10.1016/s0022-3476(83)80243-1.
2
Hypothesis: the aplastic crisis of hereditary spherocytosis is due to a single transmissible agent.假说:遗传性球形红细胞增多症的再生障碍危象是由单一可传播因子引起的。
J Clin Pathol. 1983 Apr;36(4):445-8. doi: 10.1136/jcp.36.4.445.
3
Aplastic crisis due to parvovirus infection in pyruvate kinase deficiency.丙酮酸激酶缺乏症患者因细小病毒感染引发的再生障碍危象
Lancet. 1983 Jul 2;2(8340):14-6. doi: 10.1016/s0140-6736(83)90005-3.
4
Human parvovirus, the cause of erythema infectiosum (fifth disease)?人类细小病毒,是传染性红斑(第五病)的病因吗?
Lancet. 1983 Jun 18;1(8338):1378. doi: 10.1016/s0140-6736(83)92152-9.
5
Outbreak of aplastic crises in sickle cell anaemia associated with parvovirus-like agent.与细小病毒样病原体相关的镰状细胞贫血再生障碍危象暴发。
Lancet. 1981 Sep 19;2(8247):595-7. doi: 10.1016/s0140-6736(81)92739-2.

Human parvovirus aplasia: case due to cross infection in a ward.

作者信息

Evans J P, Rossiter M A, Kumaran T O, Marsh G W, Mortimer P P

出版信息

Br Med J (Clin Res Ed). 1984 Mar 3;288(6418):681. doi: 10.1136/bmj.288.6418.681.

DOI:10.1136/bmj.288.6418.681
PMID:6320947
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1444403/
Abstract
摘要