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Transfer of demyelination by intraneural injection of experimental allergic neuritis serum.

作者信息

Saida T, Saida K, Silberberg D H, Brown M J

出版信息

Nature. 1978 Apr 13;272(5654):639-41. doi: 10.1038/272639a0.

DOI:10.1038/272639a0
PMID:643059
Abstract
摘要

相似文献

1
Transfer of demyelination by intraneural injection of experimental allergic neuritis serum.通过神经内注射实验性变应性神经炎血清传递脱髓鞘作用。
Nature. 1978 Apr 13;272(5654):639-41. doi: 10.1038/272639a0.
2
[The role of macrophage and serum factor in demyelination of experimental allergic neuritis (author's transl)].巨噬细胞和血清因子在实验性变应性神经炎脱髓鞘中的作用(作者译)
Rinsho Shinkeigaku. 1980 Nov;20(11):933-9.
3
Antiserum-mediated demyelination in vivo: a sequential study using intraneural injection of experimental allergic neuritis serum.体内抗血清介导的脱髓鞘:一项使用神经内注射实验性变应性神经炎血清的序贯研究。
Lab Invest. 1978 Nov;39(5):449-62.
4
Dominance of autoreactive T cell-mediated delayed-type hypersensitivity or antibody-mediated demyelination results in distinct forms of experimental autoimmune neuritis in the Lewis rat.自身反应性T细胞介导的迟发型超敏反应或抗体介导的脱髓鞘作用占主导地位,导致Lewis大鼠出现不同形式的实验性自身免疫性神经炎。
J Neuropathol Exp Neurol. 2001 Jun;60(6):637-46. doi: 10.1093/jnen/60.6.637.
5
Passive transfer of demyelination by experimental allergic neuritis serum.实验性变应性神经炎血清对脱髓鞘的被动转移
Acta Neuropathol. 1980;49(3):169-76. doi: 10.1007/BF00707103.
6
Presence of the terminal complement complex (C5b-9) precedes myelin degradation in immune-mediated demyelination of the rat peripheral nervous system.在大鼠外周神经系统免疫介导的脱髓鞘过程中,终末补体复合物(C5b-9)的出现先于髓鞘降解。
Ann Neurol. 1991 Aug;30(2):147-55. doi: 10.1002/ana.410300205.
7
Experimental allergic neuritis induced by sensitization with galactocerebroside.用半乳糖脑苷脂致敏诱导的实验性变应性神经炎。
Science. 1979 Jun 8;204(4397):1103-6. doi: 10.1126/science.451555.
8
Demyelinating pattern with EAE and EAN antisera studied in vitro: time lapse cinematographic analysis.体外研究EAE和EAN抗血清的脱髓鞘模式:延时电影摄影分析
Neurology. 1976 Jun;26(6 PT 2):42. doi: 10.1212/wnl.26.6_part_2.42.
9
Experimental autoimmune demyelinating diseases: experimental allergic encephalomyelitis and experimental allergic neuritis.实验性自身免疫性脱髓鞘疾病:实验性变应性脑脊髓炎和实验性变应性神经炎。
Biomedicine. 1975 Mar;22(2):81-7.
10
The occurrence of IgM and complement factors along myelin sheaths of peripheral nerves. An immunohistochemical study of the Guillain-Barré syndrome. Preliminary communication.外周神经髓鞘上IgM和补体因子的出现。格林-巴利综合征的免疫组织化学研究。初步报告。
J Neurol Sci. 1972 Feb;15(2):219-24. doi: 10.1016/0022-510x(72)90009-3.

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Puerarin accelerates neural regeneration after sciatic nerve injury.葛根素促进坐骨神经损伤后的神经再生。
Neural Regen Res. 2014 Mar 15;9(6):589-93. doi: 10.4103/1673-5374.130097.
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Propofol's effect on the sciatic nerve: Harmful or protective?丙泊酚对坐骨神经的影响:有害还是保护?
Neural Regen Res. 2013 Sep 25;8(27):2520-30. doi: 10.3969/j.issn.1673-5374.2013.27.003.
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Ursolic acid induces neural regeneration after sciatic nerve injury.熊果酸可促进坐骨神经损伤后的神经再生。
Neural Regen Res. 2013 Sep 25;8(27):2510-9. doi: 10.3969/j.issn.1673-5374.2013.27.002.
4
Animal models of autoimmune neuropathy.自身免疫性神经病的动物模型。
ILAR J. 2014;54(3):282-90. doi: 10.1093/ilar/ilt054.
5
Cross-reactivity of anti-galactocerebroside autoantibodies with a Trypanosoma brucei proteolipidic epitope.抗半乳糖脑苷脂自身抗体与布氏锥虫蛋白脂质表位的交叉反应性。
Clin Exp Immunol. 2000 Mar;119(3):516-22. doi: 10.1046/j.1365-2249.2000.01166.x.
6
Passive transfer of demyelination by experimental allergic neuritis serum.实验性变应性神经炎血清对脱髓鞘的被动转移
Acta Neuropathol. 1980;49(3):169-76. doi: 10.1007/BF00707103.
7
Effects of plasmapheresis on the course of experimental allergic neuritis in rabbits.血浆置换对兔实验性过敏性神经炎病程的影响。
J Neurol Neurosurg Psychiatry. 1981 Dec;44(12):1124-8. doi: 10.1136/jnnp.44.12.1124.
8
The treatment of inflammatory polyneuropathy by plasma exchange.血浆置换治疗炎性多发性神经病。
J Neurol Neurosurg Psychiatry. 1982 Aug;45(8):675-9. doi: 10.1136/jnnp.45.8.675.
9
Abnormal serum factors in Guillain-Barré syndrome.吉兰-巴雷综合征中的异常血清因子。
Ital J Neurol Sci. 1983 Sep;4(3):265-72. doi: 10.1007/BF02043478.
10
The effect of suppression of macrophage activity on the development of experimental allergic neuritis.巨噬细胞活性抑制对实验性变应性神经炎发展的影响。
Acta Neuropathol. 1984;62(4):316-23. doi: 10.1007/BF00687614.