Thériault G, Gingras S, Provencher S
Br J Ind Med. 1984 Aug;41(3):367-72. doi: 10.1136/oem.41.3.367.
A five step investigation was carried out to gain a better understanding of the morbidity that accompanied the development of telangiectasia on aluminium workers and to find its cause. Fifty workers with multiple telangiectasia when matched with normal controls showed the same amount of illness except that evidence of ischaemia on the ECG was found in nine cases and one control. The cases did not show an excess of abnormal biochemical tests. The basic histopathological lesion affected the surrounding tissue rather than the vessels themselves. Working in the current environment and wearing masks seems to protect young workers from developing the lesions. The Soderberg and not the prebake process was associated with the lesions; the causative agent is probably a gas that contains both hydrocarbons and fluoride components emitted from the electrolytic reactors.
开展了一项五步调查，以更好地了解铝工人毛细血管扩张症发展过程中伴随的发病率情况并找出其原因。50名患有多处毛细血管扩张症的工人与正常对照组相比，患病情况相同，只是在9例病例和1例对照中发现心电图有缺血迹象。病例组的生化检查异常情况并未增多。基本的组织病理学病变影响的是周围组织而非血管本身。在当前环境中工作并佩戴口罩似乎可保护年轻工人不发生这些病变。与这些病变相关的是索德伯格法而非预焙烧法；致病因子可能是一种含有电解反应堆排放的碳氢化合物和氟化物成分的气体。
