Behan P O
J Neurol Neurosurg Psychiatry. 1980 Jul;43(7):611-21. doi: 10.1136/jnnp.43.7.611.
In the late 1950's laboratory and clinical evidence suggested that myasthenia gravis was an autoimmune disorder. Since then a voluminous literature has developed documenting the many immunological abnormalities that occur in this condition. Recent findings point to a central disorder of immunoregulation. It is postulated that the disease occurs as a result of host genetic and environmental influences-the latter being, as yet unidentified and possibly a virus.
20世纪50年代后期，实验室和临床证据表明重症肌无力是一种自身免疫性疾病。从那时起，大量文献涌现，记录了这种疾病中出现的许多免疫异常情况。最近的研究结果指向免疫调节的核心紊乱。据推测，该病是宿主遗传和环境影响的结果——后者尚未明确，可能是一种病毒。
