母体胃泌素在先天性肥厚性幽门狭窄中重要吗？
Is maternal gastrin important in congenital hypertrophic pyloric stenosis?
作者信息
Lucey M R, McCann S, Weir D G
出版信息
Postgrad Med J. 1982 Sep;58(683):584-5. doi: 10.1136/pgmj.58.683.584.
Abstract
The birth of a normal infant to a woman with Zollinger-Ellison syndrome is described. The cord blood serum gastrin was very elevated. This suggests that maternal gastrin can cross the placenta, a fact previously in doubt in man. The infant did not develop congenital hypertrophic pyloric stenosis. This is further evidence that raised blood gastrin is not an important causative factor in the development of congenital hypertrophic pyloric stenosis in man.
摘要
本文描述了一名患有卓-艾综合征的女性产下一名正常婴儿的情况。脐带血血清胃泌素水平显著升高。这表明母体胃泌素能够穿过胎盘，这一事实在人类中此前一直存在疑问。该婴儿未患先天性肥厚性幽门狭窄。这进一步证明，血液中胃泌素水平升高并非人类先天性肥厚性幽门狭窄发生的重要致病因素。
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Nature. 1970 Jan 17;225(5229):284-5. doi: 10.1038/225284a0.
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Gastrin concentration in plasma of the neonate at birth and after the first feeding.新生儿出生时及首次喂食后血浆中胃泌素的浓度。
Pediatrics. 1976 Aug;58(2):264-7.
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Induction of pyloric hypertrophy by pentagastrin. An animal model for infantile hypertrophic pyloric stenosis.五肽胃泌素诱导幽门肥大。婴儿肥厚性幽门狭窄的动物模型。
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Increased serum immunoreactive gastrin levels in idiopathic hypertrophic pyloric stenosis.特发性肥厚性幽门狭窄患者血清免疫反应性胃泌素水平升高。
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7
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8
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