Harris C I
Biochem J. 1981 Mar 15;194(3):1011-4. doi: 10.1042/bj1941011.
The claim of Millward, Bates, Grimble, Brown, Nathan & Rennie [(1980) Biochem. J. 190. 225--228] that muscle actomyosin contributes as little as 25% of urinary N tau-methylhistidine is not consistent with other published data from that group [Bates, DeCoster, Grimble, Holloszy, Millward & Rennie (1980) J. Physiol. (London) 303, 41 P] or with literature values. It appears likely that the turnover rate of muscle actomyosin has been considerably underestimated and that when realistic rates of protein turnover are used, muscle tissue remains the major contributor of N tau-methylhistidine in urine.
米尔沃德、贝茨、格林布尔、布朗、内森和伦尼[（1980年）《生物化学杂志》190卷，225 - 228页]称肌肉肌动球蛋白对尿中N-τ-甲基组氨酸的贡献低至25%，这与该研究小组其他已发表的数据[贝茨、德科斯特、格林布尔、霍洛兹、米尔沃德和伦尼（1980年）《生理学杂志》（伦敦）303卷，41P]或文献值不一致。肌肉肌动球蛋白的周转率似乎被大大低估了，并且当使用实际的蛋白质周转率时，肌肉组织仍是尿中N-τ-甲基组氨酸的主要来源。
