Ellison R C, Sosenko J M, Harper G P, Gibbons L, Pratter F E, Miettinen O S
Hypertension. 1980 Jul-Aug;2(4 Pt 2):78-82.
It has been postulated that increased dietary sodium associated with greater food intake by obese people is a mechanism for the relationship between obesity and blood pressure (BP). We have evaluated this hypothesis by exploring the interrelationships of measures of obesity, sodium intake, and BP in 248 "normal" adolescents, 16 to 17 years of age. As an index of sodium intake, the sodium excretion in three overnight urine collections was used. As a more specific index of saltiness of diet, we used a ratio of sodium excretion to calorie intake, with calories estimated from 3-day diet records and dietary interview. Body weight and other measures of obesity showed a positive relationship with systolic blood pressure (SBP), but not with diastolic (K5) blood pressure (DBP5). Measures of overnight sodium excretion were positively correlated with body weight and calculated body fat percentage, suggesting that heavier people indeed ingest more sodium. This may result not from increased intake of food per se, but from increased saltiness of diet, since calorie intake did not increase with body weight. No significant relationships were found between BP and concurrent measures of sodium excretion or diet saltiness.
据推测，肥胖者因食物摄入量增加而导致膳食钠摄入增加，这是肥胖与血压（BP）之间关系的一种机制。我们通过研究248名16至17岁的“正常”青少年的肥胖指标、钠摄入量和血压之间的相互关系，对这一假设进行了评估。作为钠摄入量的指标，使用了三次夜间尿液收集的钠排泄量。作为饮食咸味更具体的指标，我们使用了钠排泄量与卡路里摄入量的比值，卡路里量根据3天的饮食记录和饮食访谈估算得出。体重和其他肥胖指标与收缩压（SBP）呈正相关，但与舒张压（K5）血压（DBP5）无关。夜间钠排泄量与体重和计算得出的体脂百分比呈正相关，这表明体重较重的人确实摄入了更多的钠。这可能不是由于食物摄入量本身增加，而是由于饮食咸味增加，因为卡路里摄入量并未随体重增加。未发现血压与同时测量的钠排泄量或饮食咸味之间存在显著关系。
