乙醇诱导对乙酰氨基酚的毒性及代谢。 - Suppr

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Ethanol induction of acetaminophen toxicity and metabolism.

作者信息

Peterson F J, Holloway D E, Erickson R R, Duquette P H, McClain C J, Holtzman J L

出版信息

Life Sci. 1980 Nov 3;27(18):1705-11. doi: 10.1016/0024-3205(80)90646-3.

DOI:10.1016/0024-3205(80)90646-3

PMID:7442469

Abstract

摘要

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Ethanol induction of acetaminophen toxicity and metabolism.乙醇诱导对乙酰氨基酚的毒性及代谢。

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How safe is acetaminophen?对乙酰氨基酚有多安全？

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引用本文的文献

The development and hepatotoxicity of acetaminophen: reviewing over a century of progress.对乙酰氨基酚的发展及其肝毒性：回顾一个多世纪的进展。

Drug Metab Rev. 2020 Nov;52(4):472-500. doi: 10.1080/03602532.2020.1832112. Epub 2020 Oct 14.

Paracetamol, alcohol and the liver.对乙酰氨基酚、酒精与肝脏

Br J Clin Pharmacol. 2000 Apr;49(4):291-301. doi: 10.1046/j.1365-2125.2000.00167.x.

Late presentation of acetaminophen hepatotoxicity. An unresolved problem.对乙酰氨基酚肝毒性的延迟表现。一个尚未解决的问题。

Dig Dis Sci. 1982 Apr;27(4):375-6. doi: 10.1007/BF01296761.

Paracetamol overdosage. Pharmacological considerations and clinical management.对乙酰氨基酚过量。药理学考量与临床处理

Drugs. 1983 Mar;25(3):290-314. doi: 10.2165/00003495-198325030-00002.

Lethal enhancement of therapeutic doses of acetaminophen by alcohol.酒精对治疗剂量对乙酰氨基酚的致死性增强作用。

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Immunochemical evidence for induction of the alcohol-oxidizing cytochrome P-450 of rabbit liver microsomes by diverse agents: ethanol, imidazole, trichloroethylene, acetone, pyrazole, and isoniazid.多种试剂诱导兔肝微粒体酒精氧化细胞色素P - 450的免疫化学证据：乙醇、咪唑、三氯乙烯、丙酮、吡唑和异烟肼。

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Non-narcotic analgesics. Problems of overdosage.非麻醉性镇痛药。过量用药问题。

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Glutathione deficiency in alcoholics: risk factor for paracetamol hepatotoxicity.酗酒者体内谷胱甘肽缺乏：对乙酰氨基酚肝毒性的危险因素。

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Reductive beta-scission of the hydroperoxides of fatty acids and xenobiotics: role of alcohol-inducible cytochrome P-450.脂肪酸和外源性物质氢过氧化物的还原性β-断裂：乙醇诱导的细胞色素P-450的作用

Proc Natl Acad Sci U S A. 1990 Jul;87(14):5499-503. doi: 10.1073/pnas.87.14.5499.

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