DA大鼠对油诱导性关节炎的易感性由主要组织相容性复合体（MHC）基因和非MHC基因决定。 - Suppr

Suppr

超能文献

Susceptibility to oil-induced arthritis in the DA rat is determined by MHC and non-MHC genes.

作者信息

Lorentzen J C, Olsson T, Klareskog L

机构信息

Department of Rheumatology, Karolinska Hospital, Stockholm, Sweden.

出版信息

Transplant Proc. 1995 Apr;27(2):1532-4.

PMID:7725401

Abstract

摘要

相似文献

Susceptibility to oil-induced arthritis in the DA rat is determined by MHC and non-MHC genes.DA大鼠对油诱导性关节炎的易感性由主要组织相容性复合体（MHC）基因和非MHC基因决定。

Transplant Proc. 1995 Apr;27(2):1532-4.

Modulation of multiple experimental arthritis models by collagen-induced arthritis quantitative trait loci isolated in congenic rat lines: different effects of non-major histocompatibility complex quantitative trait loci in males and females.通过在同源近交系大鼠中分离出的胶原诱导性关节炎数量性状基因座对多种实验性关节炎模型进行调控：非主要组织相容性复合体数量性状基因座在雄性和雌性中的不同作用。

Arthritis Rheum. 2002 Aug;46(8):2225-34. doi: 10.1002/art.10439.

Arthritis induced in rats with adjuvant oil is a genetically restricted, alpha beta T-cell dependent autoimmune disease.用佐剂油在大鼠中诱发的关节炎是一种受基因限制的、αβT细胞依赖性自身免疫性疾病。

Immunology. 1992 Jun;76(2):197-202.

Linkage of severity of experimental allergic encephalomyelitis to the rat major histocompatibility locus.实验性变应性脑脊髓炎的严重程度与大鼠主要组织相容性位点的连锁关系。

J Immunol. 1980 Apr;124(4):1815-20.

Association between susceptibility to experimental allergic encephalomyelitis and the major histocompatibility system in congenic rat strains.近交系大鼠品系中实验性变应性脑脊髓炎易感性与主要组织相容性系统之间的关联。

Clin Exp Immunol. 1978 Jun;32(3):429-34.

Susceptibility of DA rats to arthritis induced with adjuvant oil or rat collagen is determined by genes both within and outside the major histocompatibility complex.DA大鼠对佐剂油或大鼠胶原蛋白诱导的关节炎的易感性由主要组织相容性复合体内外的基因决定。

Scand J Immunol. 1996 Dec;44(6):592-8. doi: 10.1046/j.1365-3083.1996.d01-354.x.

Detection of arthritis-susceptibility loci, including Ncf1, and variable effects of the major histocompatibility complex region depending on genetic background in rats.大鼠中关节炎易感基因座（包括Ncf1）的检测以及主要组织相容性复合体区域因遗传背景而异的可变效应。

Arthritis Rheum. 2009 Feb;60(2):419-27. doi: 10.1002/art.24292.

High resolution mapping of an arthritis susceptibility locus on rat chromosome 4, and characterization of regulated phenotypes.大鼠4号染色体上关节炎易感基因座的高分辨率定位及调控表型的特征分析。

Hum Mol Genet. 2003 Sep 1;12(17):2087-96. doi: 10.1093/hmg/ddg224. Epub 2003 Jul 8.

Relationship between collagen-induced and adjuvant arthritis in the Lewis rat.胶原诱导性关节炎与佐剂性关节炎在Lewis大鼠中的关系。

J Autoimmun. 1993 Dec;6(6):691-700. doi: 10.1006/jaut.1993.1058.

Resistance and susceptibility to the induction of rat adjuvant disease. Diverging susceptibility and severity achieved by selective breeding.大鼠佐剂病诱导的抗性与易感性。通过选择性育种实现的不同易感性和严重程度。

Br J Exp Pathol. 1979 Oct;60(5):507-12.

引用本文的文献

Genes and environment in arthritis: can RA be prevented?关节炎中的基因与环境：类风湿关节炎能否预防？

Arthritis Res. 2002;4 Suppl 3(Suppl 3):S31-6. doi: 10.1186/ar566. Epub 2002 May 9.

Identification of rat susceptibility loci for adjuvant-oil-induced arthritis.鉴定佐剂油诱导性关节炎大鼠的易感性基因座。

Proc Natl Acad Sci U S A. 1998 May 26;95(11):6383-7. doi: 10.1073/pnas.95.11.6383.

Common commercial cosmetic products induce arthritis in the DA rat.常见的商业化妆品会诱发DA大鼠患关节炎。

Environ Health Perspect. 1998 Jan;106(1):27-32. doi: 10.1289/ehp.9810627.

Polyclonal Th1 cells transfer oil-induced arthritis.多克隆Th1细胞可转移油诱导的关节炎。

Immunology. 1997 Jun;91(2):260-5. doi: 10.1046/j.1365-2567.1997.00251.x.

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